Correction to “The SWI/SNF ATPases are required for triple negative breast cancer cell proliferation”

IF 4 2区 生物学 Q2 CELL BIOLOGY Journal of Cellular Physiology Pub Date : 2024-08-27 DOI:10.1002/jcp.31395
{"title":"Correction to “The SWI/SNF ATPases are required for triple negative breast cancer cell proliferation”","authors":"","doi":"10.1002/jcp.31395","DOIUrl":null,"url":null,"abstract":"<p>Wu, Q., Madany, P., Akech, J., Dobson, J.R., Douthwright, S., Browne, G., Colby, J.L., Winter, G.E., Bradner, J.E., Pratap, J., Sluder, G., Bhargava, R., Chiosea, S.I., van Wijnen, A.J., Stein, J.L., Stein, G.S., Lian, J.B., Nickerson, J.A. and Imbalzano, A.N. (2015), The SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell Proliferation. <i>J. Cell. Physiol</i>., 230: 2683-2694. https://doi.org/10.1002/jcp.24991</p><p>In the original version of this article, the authors mistakenly duplicated panels across Figure 5B (“Scram” and “BRM KD”) during the selection and assembly of the images. The correct Figure 5B is presented below, where the “Scram” panel has been replaced.</p><p>Additionally, it was noted that the Western Blot image for BRG1 in Figure 3C contains an apparent splice site. The authors have provided data from a replicate experiment to address the issue. The corrected Figure 3C is shown below.</p><p>This correction doesn't change the results and conclusions. The authors apologize for any confusion these errors may have caused.</p>","PeriodicalId":15220,"journal":{"name":"Journal of Cellular Physiology","volume":"239 11","pages":""},"PeriodicalIF":4.0000,"publicationDate":"2024-08-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1002/jcp.31395","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of Cellular Physiology","FirstCategoryId":"99","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1002/jcp.31395","RegionNum":2,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q2","JCRName":"CELL BIOLOGY","Score":null,"Total":0}
引用次数: 0

Abstract

Wu, Q., Madany, P., Akech, J., Dobson, J.R., Douthwright, S., Browne, G., Colby, J.L., Winter, G.E., Bradner, J.E., Pratap, J., Sluder, G., Bhargava, R., Chiosea, S.I., van Wijnen, A.J., Stein, J.L., Stein, G.S., Lian, J.B., Nickerson, J.A. and Imbalzano, A.N. (2015), The SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell Proliferation. J. Cell. Physiol., 230: 2683-2694. https://doi.org/10.1002/jcp.24991

In the original version of this article, the authors mistakenly duplicated panels across Figure 5B (“Scram” and “BRM KD”) during the selection and assembly of the images. The correct Figure 5B is presented below, where the “Scram” panel has been replaced.

Additionally, it was noted that the Western Blot image for BRG1 in Figure 3C contains an apparent splice site. The authors have provided data from a replicate experiment to address the issue. The corrected Figure 3C is shown below.

This correction doesn't change the results and conclusions. The authors apologize for any confusion these errors may have caused.

Abstract Image

查看原文
分享 分享
微信好友 朋友圈 QQ好友 复制链接
本刊更多论文
对 "三阴性乳腺癌细胞增殖需要 SWI/SNF ATPases "的更正。
Wu, Q., Madany, P., Akech, J., Dobson, J.R., Douthwright, S., Browne, G., Colby, J.L., Winter, G.E., Bradner, J.E., Pratap, J., Sluder, G., Bhargava, R., Chiosea, S.I., van Wijnen, A.J., Stein, J.L., Stein, G.S., Lian, J.B., Nickerson, J.A. and Imbalzano, A.J..、van Wijnen, A.J., Stein, J.L., Stein, G.S., Lian, J.B., Nickerson, J.A. and Imbalzano, A.N. (2015), The SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell Proliferation.J. Cell.Physiol:https://doi.org/10.1002/jcp.24991In 在本文的原始版本中,作者在选择和组合图像时错误地重复了图 5B 的面板("Scram "和 "BRM KD")。下面是正确的图 5B,其中的 "Scram "面板已被替换。此外,有人指出图 3C 中 BRG1 的 Western 印迹图像包含一个明显的剪接位点。作者提供了重复实验的数据来解决这个问题。更正后的图 3C 如下所示。作者对这些错误可能造成的混淆表示歉意。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
求助全文
约1分钟内获得全文 去求助
来源期刊
CiteScore
14.70
自引率
0.00%
发文量
256
审稿时长
1 months
期刊介绍: The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
期刊最新文献
Role of Chitinase 3-like-1 in Myelofibrosis via Fibroblast-Produced Extracellular Matrix Enhancement. Impact of Maternal Supraphysiological Hypercholesterolemia on Lysosomal and Mitochondrial Function in Placental Trophoblast Cells. Innate Immune Receptor NLRX1: Potential Modulator of Glioblastoma Pathophysiology The Laminin-Derived Peptide C16 Interacts With β1 Integrin, and Is Internalized by Vesicles Through Endocytic Pathway Correction to “A Negative Feedback Loop of H19/miR-675/VDR Mediates Therapeutic Effect of Curcumin in the Treatment of Glioma”
×
引用
GB/T 7714-2015
复制
MLA
复制
APA
复制
导出至
BibTeX EndNote RefMan NoteFirst NoteExpress
×
×
提示
您的信息不完整,为了账户安全,请先补充。
现在去补充
×
提示
您因"违规操作"
具体请查看互助需知
我知道了
×
提示
现在去查看 取消
×
提示
确定
0
微信
客服QQ
Book学术公众号 扫码关注我们
反馈
×
意见反馈
请填写您的意见或建议
请填写您的手机或邮箱
已复制链接
已复制链接
快去分享给好友吧!
我知道了
×
扫码分享
扫码分享
Book学术官方微信
Book学术文献互助
Book学术文献互助群
群 号:604180095
Book学术
文献互助 智能选刊 最新文献 互助须知 联系我们:info@booksci.cn
Book学术提供免费学术资源搜索服务,方便国内外学者检索中英文文献。致力于提供最便捷和优质的服务体验。
Copyright © 2023 Book学术 All rights reserved.
ghs 京公网安备 11010802042870号 京ICP备2023020795号-1