Corrigendum: Vasorin as an actor of bone turnover?

IF 4 2区生物学 Q2 CELL BIOLOGY Journal of Cellular Physiology Pub Date : 2024-08-27 DOI:10.1002/jcp.31425

引用次数: 0

Abstract

This article corrects the following:

Vasorin as an actor of bone turnover?

Caroline Andrique, Anne Laure Bonnet, Julien Dang, Julie Lesieur, A. Michaela Krautzberger, Brigitte Baroukh, Coralie Torrens, Jeremy Sadoine, Alain Schmitt, Gael Y. Rochefort, Claire Bardet, Isabelle Six, Pascal Houillier, Pierre Louis Tharaux, Heinrich Schrewe, Celine Gaucher, Catherine Chaussain

Volume 239, Issue 6, Journal of Cellular Physiology

https://doi.org/10.1002/jcp.31257

First published online: 12 June 2024

In the original version of the article, an author was inadvertently omitted from the author list.

In the corrected article, M Eijken¹⁰, has been added between Baroukh B¹ and Torrens C¹.

In addition, “10. department of clinical medicine, Aarhus university, 8200 Aarhus N, Denmark, department of renal medicine, Aarhus university hospital, 8200 Aarhus N, Denmark.” Has been added to the affiliations list.

The authors apologize for the error.

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更正：瓦索林（Vasorin）是骨转换的促进剂？

本文纠正以下错误：Vasorin是骨转换的一个参与者？Caroline Andrique, Anne Laure Bonnet, Julien Dang, Julie Lesieur， A. Michaela Krautzberger, Brigitte Baroukh, Coralie Torrens, Jeremy Sadoine, Alain Schmitt, Gael Y. Rochefort, Claire Bardet, Isabelle Six, Pascal Houillier, Pierre Louis Tharaux, Heinrich Schrewe, Celine Gaucher， Catherine chaussain第239卷第6期《细胞生理学杂志》https://doi.org/10.1002/jcp.31257First在线发表：在文章的原始版本中，作者列表中无意中遗漏了一位作者。在更正后的文章中，在Baroukh B1和Torrens C1之间添加了M Eijken10。此外，“10.”丹麦奥胡斯大学临床医学系，丹麦奥胡斯大学附属医院肾脏医学系，丹麦奥胡斯大学附属医院，8200已添加到从属关系列表中。作者为这个错误道歉。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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来源期刊

Journal of Cellular Physiology 生物-生理学

CiteScore

14.70

自引率

0.00%

发文量

256

审稿时长

1 months

期刊介绍： The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.