RETRACTION

IF 4.5 2区 生物学 Q2 CELL BIOLOGY Journal of Cellular Physiology Pub Date : 2024-09-30 DOI:10.1002/jcp.31392
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Abstract

RETRACTION: M. Du, Y. Zhuang, P. Tan, Z. Yu, X. Zhang, A. Wang, “MicroRNA-95 Knockdown Inhibits Epithelial–mesenchymal Transition and Cancer Stem Cell Phenotype in Gastric Cancer Cells through MAPK Pathway by Upregulating DUSP5,” Journal of Cellular Physiology 235, no. 2 (2020): 944-956, https://doi.org/10.1002/jcp.29010.

The above article, published online on 15 July 2019 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Robert Heath; and Wiley Periodicals LLC. The retraction has been agreed due to concerns raised by third parties on the data presented in the article. Specifically, the article makes use of unverifiable/unknown and/or contaminated cell lines. Additionally, the description of experimental methods is insufficient and relevant supporting data is unavailable, making the foundation of the study not comprehensible and/or reproducible to readers. Accordingly, the conclusions of this article are considered invalid by the editors. The authors have been informed of the decision of retraction but unavailable for a final confirmation.

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撤回:M. Du, Y. Zhuang, P. Tan, Z. Yu, X. Zhang, A. Wang, "MicroRNA-95 Knockdown Inhibits Epithelial-mesenchymal Transition and Cancer Stem Cell Phenotype in Gastric Cancer Cells through MAPK Pathway by Upregulating DUSP5," Journal of Cellular Physiology 235, no. 2 (2020): 944-956, https://doi.org/10.1002/jcp.29010.上述文章于 2019 年 7 月 15 日在线发表于 Wiley Online Library (wileyonlinelibrary.com),经期刊主编 Robert Heath 和 Wiley Periodicals LLC 协议,该文章已被撤回。同意撤稿的原因是第三方对文章中提供的数据表示担忧。具体来说,文章使用了无法验证/未知和/或受污染的细胞系。此外,文章对实验方法的描述不够充分,也没有提供相关的支持数据,使得读者无法理解和/或复制该研究的基础。因此,编辑认为这篇文章的结论无效。作者已被告知撤稿决定,但无法得到最终确认。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
CiteScore
14.70
自引率
0.00%
发文量
256
审稿时长
1 months
期刊介绍: The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
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