Corrigendum: RA promotes proliferation of primordial germ cell-like cells differentiated from porcine skin-derived stem cells

IF 4.5 2区生物学 Q2 CELL BIOLOGY Journal of Cellular Physiology Pub Date : 2024-10-14 DOI:10.1002/jcp.31470

引用次数: 0

Abstract

This article corrects the following:

RA promotes proliferation of primordial germ cell-like cells differentiated from porcine skin-derived stem cells.

Hong-Chen Yan, Lin Li, Jing-Cai Liu, Yu-Feng Wang, Xue-Lian Liu, Wei Ge, Paul W. Dyce, Lan Li, Xiao-Feng Sun, Wei Shen, Shun-Feng Cheng

Volume 234, Issue 10, Journal of Cellular Physiology

https://doi.org/10.1002/jcp.28454

First published online: 11 March 2019

Volume/Issue/Pages: Journal of Cellular Physiology; 2019; 234: 18214-18229.

Correction text:

The authors recently found an error in Figure 8a in the original article, wherein an incorrect fluorescence image of Vasa staining was used. This error occurred accidentally during the selection of the representative images. The alteration does not affect the results or conclusions of Figure 8. The authors regret their error and apologize for any inconvenience to readers arising from the error.

The corrected figure is displayed below.

Abstract Image

查看原文

微信好友朋友圈 QQ好友复制链接

本刊更多论文

更正：RA可促进由猪皮肤干细胞分化而来的原始生殖细胞样细胞的增殖。

本文更正以下内容：RA促进从猪皮肤干细胞分化的原始生殖细胞样细胞的增殖。闫洪辰，李林，刘景才，王玉峰，刘雪莲，葛伟，Paul W. Dyce，李兰，孙晓峰，沈伟，程顺峰，第234卷，第10期，《细胞生理学杂志》https://doi.org/10.1002/jcp.28454First在线出版：2019年3月11日卷/期/页：《细胞生理学杂志》；2019年;234: 18214 - 18229。更正文本：作者最近在原文的图8a中发现了一个错误，其中使用了错误的Vasa染色的荧光图像。此错误是在选择代表性图像时意外发生的。更改不会影响图8的结果或结论。作者对错误表示歉意，并对由此给读者带来的不便表示歉意。更正后的数字如下所示。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

求助全文

约1分钟内获得全文去求助

来源期刊

Journal of Cellular Physiology 生物-生理学

CiteScore

14.70

自引率

0.00%

发文量

256

审稿时长

1 months

期刊介绍： The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.