The relation between adverse experiences and proactive aggression: The moderating role of emotional sensitivity

IF 3.5 2区 心理学 Q1 PSYCHOLOGY, SOCIAL Personality and Individual Differences Pub Date : 2024-11-05 DOI:10.1016/j.paid.2024.112944
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Abstract

Although adverse experiences might influence human aggressive behavior, not all individuals who experience adverse experiences will behave aggressively. Therefore, it is necessary to study the moderating mechanisms underlying this relationship. Based on the diathesis-stress model, we explored the moderating role of emotional sensitivity in the association between adverse experiences and proactive aggressive behavior. Specifically, 940 individuals completed the measures of adverse experiences (family neglect and community violence exposure), proactive aggression and emotional sensitivity. The results found that adverse experiences were significantly and positively associated with one's aggressive behaviors. Furthermore, the strength of these relationships was moderated by emotional sensitivity. Individuals with low emotional sensitivity showed weaker associations compared to those with high emotional sensitivity, suggesting the lower emotional sensitivity served as a buffer against the negative effects of adverse experiences on proactive aggressive behavior. These findings supported the diathesis-stress model and deepen our understanding of the link between adverse experiences and proactive aggression.
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不良经历与主动攻击之间的关系:情绪敏感性的调节作用
尽管不良经历可能会影响人类的攻击性行为,但并非所有经历过不良经历的个体都会有攻击性行为。因此,有必要研究这种关系的调节机制。我们基于 "病因-压力 "模型,探讨了情绪敏感性在不良经历与主动攻击行为之间的调节作用。具体而言,940 人完成了不良经历(家庭忽视和社区暴力暴露)、主动攻击行为和情绪敏感性的测量。结果发现,不良经历与个人的攻击行为有显著的正相关关系。此外,这些关系的强度受情绪敏感性的调节。与情绪敏感度高的人相比,情绪敏感度低的人的相关性较弱,这表明情绪敏感度低的人可以缓冲不良经历对主动攻击行为的负面影响。这些研究结果支持了 "压力模式",并加深了我们对不良经历与主动攻击行为之间联系的理解。
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来源期刊
CiteScore
8.50
自引率
4.70%
发文量
577
审稿时长
41 days
期刊介绍: Personality and Individual Differences is devoted to the publication of articles (experimental, theoretical, review) which aim to integrate as far as possible the major factors of personality with empirical paradigms from experimental, physiological, animal, clinical, educational, criminological or industrial psychology or to seek an explanation for the causes and major determinants of individual differences in concepts derived from these disciplines. The editors are concerned with both genetic and environmental causes, and they are particularly interested in possible interaction effects.
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