Author Correction: Brca1 haploinsufficiency promotes early tumor onset and epigenetic alterations in a mouse model of hereditary breast cancer

IF 31.7 1区 生物学 Q1 GENETICS & HEREDITY Nature genetics Pub Date : 2024-11-20 DOI:10.1038/s41588-024-02034-9
Carman Man-Chung Li, Alyssa Cordes, Michael U. J. Oliphant, S. Aidan Quinn, Mayura Thomas, Laura M. Selfors, Francesca Silvestri, Nomeda Girnius, Gianmarco Rinaldi, Jason J. Zoeller, Hana Shapiro, Christina Tsiobikas, Kushali P. Gupta, Shailja Pathania, Aviv Regev, Cigall Kadoch, Senthil K. Muthuswamy, Joan S. Brugge
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引用次数: 0

Abstract

Correction to: Nature Genetics https://doi.org/10.1038/s41588-024-01958-6, published online 11 November 2024.

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作者更正:在遗传性乳腺癌小鼠模型中,Brca1单倍体缺失会促进肿瘤的早期发生和表观遗传学改变
更正为自然遗传学》https://doi.org/10.1038/s41588-024-01958-6,2024 年 11 月 11 日在线发表。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
Nature genetics
Nature genetics 生物-遗传学
CiteScore
43.00
自引率
2.60%
发文量
241
审稿时长
3 months
期刊介绍: Nature Genetics publishes the very highest quality research in genetics. It encompasses genetic and functional genomic studies on human and plant traits and on other model organisms. Current emphasis is on the genetic basis for common and complex diseases and on the functional mechanism, architecture and evolution of gene networks, studied by experimental perturbation. Integrative genetic topics comprise, but are not limited to: -Genes in the pathology of human disease -Molecular analysis of simple and complex genetic traits -Cancer genetics -Agricultural genomics -Developmental genetics -Regulatory variation in gene expression -Strategies and technologies for extracting function from genomic data -Pharmacological genomics -Genome evolution
期刊最新文献
Author Correction: Brca1 haploinsufficiency promotes early tumor onset and epigenetic alterations in a mouse model of hereditary breast cancer Epigenetic scars of Brca1 loss point toward breast cancer cell of origin A multilineage screen identifies actionable synthetic lethal interactions in human cancers Intermediate cells with activated JAK/STAT signaling in prostate regeneration and diseases Toward GDPR compliance with the Helmholtz Munich genotype imputation server
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