Lateral Femoral Notch Sign Presence, Location, and Depth Are Not Associated with Primary Anterior Cruciate Ligament Reconstruction Failure: A Retrospective Case-Control Study.
Anna M Ifarraguerri, Michael S Collins, Ainsley K Bloomer, Kennedy K Gachigi, David Trofa, Patrick Siparsky, Dana P Piasecki, James E Fleischli, Bryan M Saltzman
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引用次数: 0
Abstract
Purpose: The purpose of this study is to assess the relationship between the presence, depth, and location of the lateral femoral notch sign (LFNS) on preoperative MRI and the risk of ACL reconstruction (ACLR) graft failure, as well as secondary return to sport (RTS) endpoints.
Methods: Patients with primary ACLR failure between 2012 and 2021 with a minimum of a 2-year follow-up were identified and matched to patients without primary ACLR failure by sex, age, and BMI. Patients with incomplete medical records or concomitant lateral extra-articular tenodesis or anterolateral ligament reconstruction were excluded. The LFNS presence, depth and location were recorded from patients' preoperative MRI. Intraoperative data, concomitant injuries, ACLR failure, and return to sport (RTS) were collected.
Results: Of the 253 included patients, 158(62.5%) were male, the mean age was 22 ± 9.1 years old, and the mean body mass index (BMI) was 25.7 ± 5.7 kg/m2. 87(34.4%) had a LFNS on preoperative MRI. There was no difference in the prevalence of the LFNS between patients with primary ACLR failure (42(32.1%)) and without primary ACLR failure (45(36.9%)) (OR 1.24, 95% CI 0.74 to 2.08; P=0.42). Among patients with the LFNS, there was no difference in mean depth between those with and without primary ACLR failure, or when stratifying depth by 1.0-1.5mm, 1.5-2.0mm, and >2.0mm. The mean location of the LFNS from Blumensaat line did not differ between patients with or without primary ACLR failure, and RTS rate, level, and time were comparable between patients with and without the LFNS.
Conclusion: There was no significant difference in the presence, depth or location of LFNS in patients with and without primary ACLR failure. Presence of the LFNS is not associated with additional risk of primary ACLR failure, and clinical outcomes were comparable in patients with and without the LFNS.
Level of evidence: Retrospective case-control study; IV.
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