{"title":"Correction to “TAK1 mediates excessive autophagy via p38 and ERK in cisplatin-induced acute kidney injury”","authors":"","doi":"10.1111/jcmm.70414","DOIUrl":null,"url":null,"abstract":"<p>Zhou J, Fan Y, Zhong J, et al. TAK1 mediates excessive autophagy via p38 and ERK in cisplatin-induced acute kidney injury. <i>Journal of cellular and molecular medicine</i> 2018;22(5):2908–2921. doi:10.1111/jcmm.13585.</p><p>In Zhou Jun et al., the immunohistochemistry (IHC) image for sham in Figure 1H was inadvertently misused during the figure preparation process. This correction does not affect the figure legends or the conclusions drawn in the study. The correct figures are shown below.</p>","PeriodicalId":101321,"journal":{"name":"JOURNAL OF CELLULAR AND MOLECULAR MEDICINE","volume":"29 3","pages":""},"PeriodicalIF":4.2000,"publicationDate":"2025-02-12","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/jcmm.70414","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"JOURNAL OF CELLULAR AND MOLECULAR MEDICINE","FirstCategoryId":"1085","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1111/jcmm.70414","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 0
Abstract
Zhou J, Fan Y, Zhong J, et al. TAK1 mediates excessive autophagy via p38 and ERK in cisplatin-induced acute kidney injury. Journal of cellular and molecular medicine 2018;22(5):2908–2921. doi:10.1111/jcmm.13585.
In Zhou Jun et al., the immunohistochemistry (IHC) image for sham in Figure 1H was inadvertently misused during the figure preparation process. This correction does not affect the figure legends or the conclusions drawn in the study. The correct figures are shown below.
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The Journal of Cellular and Molecular Medicine serves as a bridge between physiology and cellular medicine, as well as molecular biology and molecular therapeutics. With a 20-year history, the journal adopts an interdisciplinary approach to showcase innovative discoveries.
It publishes research aimed at advancing the collective understanding of the cellular and molecular mechanisms underlying diseases. The journal emphasizes translational studies that translate this knowledge into therapeutic strategies. Being fully open access, the journal is accessible to all readers.
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