RETRACTION: MiR-101-3p Regulates the Viability of Lung Squamous Carcinoma Cells via Targeting EZH2

IF 2.8 3区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY Journal of cellular biochemistry Pub Date : 2025-02-25 DOI:10.1002/jcb.70007
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引用次数: 0

Abstract

RETRACTION: Y. Hou, L. Li, Y. Ju, Y. Lu, L. Chang, and X. Xiang, “MiR-101-3p Regulates the Viability of Lung Squamous Carcinoma Cells via Targeting EZH2,” Journal of Cellular Biochemistry 118, no. 10 (2017): 3142-3149, https://doi.org/10.1002/jcb.25836.

The above article, published online on 14 December 2016 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the authors; the journal Editor-in-Chief, Christian Behl; and Wiley Periodicals LLC. The retraction has been agreed due to concerns raised by third parties on the data presented in the article. Specifically, duplication of multiple image elements within Figure 3 C and 3E has been identified. Additional flaws and inconsistencies between methodology described and results presented were found. The authors were not able to provide comprehensive raw data. Accordingly, the article is retracted as the editors have lost confidence in the integrity and reliability of the full body of data presented in the article and consider its conclusions invalid.

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撤回:MiR-101-3p通过靶向EZH2调控肺鳞癌细胞活力
引用本文:侯艳,李丽,朱艳,卢艳,张丽,向翔,“MiR-101-3p对肺鳞癌细胞生存能力的调控作用”,《细胞生物化学》第18期,第1期。10 (2017): 3142-3149, https://doi.org/10.1002/jcb.25836.The上述文章于2016年12月14日在线发表在Wiley online Library (wileyonlinelibrary.com),经作者同意撤回;杂志主编克里斯蒂安·贝尔;和Wiley期刊有限责任公司。由于第三方对文章中提供的数据提出了担忧,已经同意撤回。具体来说,图3c和图3E中存在多个图像元素的重复。在描述的方法和提出的结果之间发现了额外的缺陷和不一致。作者无法提供全面的原始数据。因此,由于编辑对文章中提供的全部数据的完整性和可靠性失去信心,并认为其结论无效,因此文章被撤回。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
Journal of cellular biochemistry
Journal of cellular biochemistry 生物-生化与分子生物学
CiteScore
9.90
自引率
0.00%
发文量
164
审稿时长
1 months
期刊介绍: The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.
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