{"title":"Corrigendum to “The Effects and Mechanisms of Myeloid Differentiation Protein 2 on Intestinal Mucosal Permeability in Mice With Chronic Colitis”","authors":"","doi":"10.1002/jcp.70009","DOIUrl":null,"url":null,"abstract":"<p>Han C., Q. Guan, L. Guo, Y. Yang, S. Ruan, and X. Zhang. 2019. “The Effects and Mechanisms of Myeloid Differentiation Protein 2 on Intestinal Mucosal Permeability in Mice With Chronic Colitis.” <i>Journal of Cellular Physiology</i> 234, no. 11: 21089–21099. https://doi.org/10.1002/jcp.28711.</p><p>In the initial version of the proof stage, We mistakenly inserted incorrect panels in Figure 2d, Figure 3a, Figure 4d, Figure 5a, and Figure 6c. Specifically:</p><p>Figure 2c: We have confused the panels of Con and DSS, and the magnification is not consistent. We will present the correct results as follows:</p><p>Figure 3a: Strip insertion error for MD2. We will present the correct results as follows:</p><p>Figure 4d: TLR4 strip insertion error. We will present the correct results as follows:</p><p>Figure 5a: MD2 and GAPDH strip insertion error. We will present the correct results as follows:</p><p>Figure 6c: GAPDH strip insertion error. We will present the correct results as follows:</p><p>This correction doesn't change the results and conclusions. The authors apologize for any confusion these errors may have caused.</p>","PeriodicalId":15220,"journal":{"name":"Journal of Cellular Physiology","volume":"240 2","pages":""},"PeriodicalIF":4.5000,"publicationDate":"2025-02-26","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1002/jcp.70009","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of Cellular Physiology","FirstCategoryId":"99","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1002/jcp.70009","RegionNum":2,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q2","JCRName":"CELL BIOLOGY","Score":null,"Total":0}
引用次数: 0
Abstract
Han C., Q. Guan, L. Guo, Y. Yang, S. Ruan, and X. Zhang. 2019. “The Effects and Mechanisms of Myeloid Differentiation Protein 2 on Intestinal Mucosal Permeability in Mice With Chronic Colitis.” Journal of Cellular Physiology 234, no. 11: 21089–21099. https://doi.org/10.1002/jcp.28711.
In the initial version of the proof stage, We mistakenly inserted incorrect panels in Figure 2d, Figure 3a, Figure 4d, Figure 5a, and Figure 6c. Specifically:
Figure 2c: We have confused the panels of Con and DSS, and the magnification is not consistent. We will present the correct results as follows:
Figure 3a: Strip insertion error for MD2. We will present the correct results as follows:
Figure 4d: TLR4 strip insertion error. We will present the correct results as follows:
Figure 5a: MD2 and GAPDH strip insertion error. We will present the correct results as follows:
Figure 6c: GAPDH strip insertion error. We will present the correct results as follows:
This correction doesn't change the results and conclusions. The authors apologize for any confusion these errors may have caused.
期刊介绍:
The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.