Correction to “Overexpression of lncRNA WWTR1-AS1 Associates With Tumor Aggressiveness and Unfavorable Survival in Head–Neck Squamous Cell Carcinoma”

IF 3 3区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY Journal of cellular biochemistry Pub Date : 2025-03-02 DOI:10.1002/jcb.70004
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Abstract

J. Li, Z. Li, Y. Wu, et al., “Overexpression of lncRNA WWTR1-AS1 Associates With Tumor Aggressiveness and Unfavorable Survival in Head–Neck Squamous Cell Carcinoma,” Journal of Cellular Biochemistry 120, no. 10 (2019): 18266–18277, https://doi.org/10.1002/jcb.29132.

In the published article, the representative flow cytometry plot in Figure 3C (NC Cal27) was included by mistake during figure preparation. The authors have corrected this issue by replacing this image with the correct original flow cytometry plot. The corrected Figure 3 is shown below.

The authors also found that the GAPDH blots in Figure 4D were inadvertently duplicated (Cal27 and FaDu cells treated with either control-ASO or WWTR1-AS1-ASO-393). Based on the original blots, the authors confirm that the GAPDH blot for FaDu cells is correct, while the blot for Cal27 cells has been included by mistake. The corrected Figure 4 is shown below.

The authors apologize for these errors and for any inconvenience these may have caused.

Abstract Image

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来源期刊
Journal of cellular biochemistry
Journal of cellular biochemistry 生物-生化与分子生物学
CiteScore
9.90
自引率
0.00%
发文量
164
审稿时长
1 months
期刊介绍: The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.
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