Stuttering and phonological disorders in children: examination of the Covert Repair Hypothesis.

J S Yaruss, E G Conture
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引用次数: 65

Abstract

The purpose of this study was to evaluate whether the Covert Repair Hypothesis (CRH; Postma & Kolk, 1993), a theory designed to account for the occurrence of speech disfluencies in adults who stutter, can also account for selected speech characteristics of children who stutter and demonstrate disordered phonology. Subjects were 9 boys who stutter and exhibit normal phonology (S + NP; mean age = 61.33 months; SD = 10.16 months) and 9 boys who stutter and exhibit disordered phonology (S + DP; mean age = 59.11 months; SD = 9.37 months). Selected aspects of each child's speech fluency and phonology were analyzed on the basis of an audio/videotaped picture-naming task and a 30-min conversational interaction with his mother. Results indicated that S + NP and S + DP children are generally comparable in terms of their basic speech disfluency, nonsystematic speech error, and self-repair behaviors. CRH predictions that utterances produced with faster articulatory speaking rates or shorter response time latencies are more likely to contain speech errors or speech disfluencies were not supported. CRH predictions regarding the co-occurrence of speech disfluencies and speech errors were supported for nonsystematic ("slip-of-the-tongue"), but not for systematic (phonological process/rule-bases), speech errors. Furthermore, neither S + NP nor S + DP subjects repaired their systematic speech errors during conversational speech, suggesting that systematic deviations from adult forms may not represent true "errors, " at least for some children exhibiting phonological processes. Findings suggest that speech disfluencies may not represent by-products of self-repairs of systematic speech errors produced during conversational speech, but that self-repairs of nonsystematic speech errors may be related to children's production of speech disfluencies.

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儿童口吃和语音障碍:隐性修复假说的检验。
本研究的目的是评估隐性修复假说(CRH;Postma & Kolk, 1993),一个用来解释成人口吃中言语不流利的理论,也可以解释口吃和表现出语音紊乱的儿童的特定言语特征。研究对象为9名口吃且语音正常的男孩(S + NP;平均年龄61.33个月;SD = 10.16个月)和9名口吃和表现出语音障碍的男孩(S + DP;平均年龄59.11个月;SD = 9.37个月)。每个孩子的语言流畅性和音韵学的选择方面进行分析的基础上,音频/录像的图片命名任务和30分钟的对话互动与他的母亲。结果表明,S + NP儿童和S + DP儿童在基本言语不流利、非系统言语错误和自我修复行为方面基本具有可比性。CRH预测,以更快的发音速度或更短的反应时间延迟产生的话语更有可能包含言语错误或言语不流利,这一预测不受支持。CRH对非系统性(“口误”)语音不流利和语音错误共同发生的预测得到支持,但对系统性(语音过程/规则基础)语音错误则不支持。此外,S + NP和S + DP被试在会话言语中都没有修复他们的系统性言语错误,这表明与成人形式的系统性偏差可能并不代表真正的“错误”,至少对一些表现出语音过程的儿童来说是这样。研究结果表明,言语不流利可能不是会话言语中产生的系统性言语错误自我修复的副产品,但非系统性言语错误的自我修复可能与儿童言语不流利的产生有关。
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