{"title":"Pneumothorax complicating laparoscopic ureterolysis.","authors":"S Altarac, G Janetschek, E Eder, G Bartsch","doi":"10.1089/lps.1996.6.193","DOIUrl":null,"url":null,"abstract":"<p><p>In a 71-year-old female marked left-sided ureteral stenosis secondary to retroperitoneal fibrosis was diagnosed. Since conservative therapy with cortisone had failed, laparoscopic ureterolysis was performed. Following tracheal intubation the lungs were ventilated with 40 vol% O2 in air and isoflurane 0.5-2%, using a positive end-expiratory pressure of 6 cm H2O. A CO2 pneumoperitoneum was established with a pressure-controlled high-flow insufflator; the intraabdominal pressure during the procedure was 14 mm Hg. Two hours after gas instillation, the peak airway pressure increased from 22 to 40 cm H2O, and the PaCO2 from 45 to 70 mm Hg. Breath sounds over the right lung were no longer heard, and subcutaneous emphysema was noted over the neck and face. An intraoperative chest X-ray confirmed a right pneumothorax. Following peritoneal gas evacuation, the PaCO2 returned to 35 mm Hg, the subcutaneous emphysema diminished, and a repeat chest X-ray showed complete resolution of the pneumothorax. The course of this event led us to the conclusion that the pneumothorax was due to diffusion of CO2 from the peritoneal to the pleural cavity through congenital defects in the diaphragm. Ureterolysis could be continued by laparotomy.</p>","PeriodicalId":77211,"journal":{"name":"Journal of laparoendoscopic surgery","volume":"6 3","pages":"193-6"},"PeriodicalIF":0.0000,"publicationDate":"1996-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1089/lps.1996.6.193","citationCount":"17","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of laparoendoscopic surgery","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.1089/lps.1996.6.193","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 17
Abstract
In a 71-year-old female marked left-sided ureteral stenosis secondary to retroperitoneal fibrosis was diagnosed. Since conservative therapy with cortisone had failed, laparoscopic ureterolysis was performed. Following tracheal intubation the lungs were ventilated with 40 vol% O2 in air and isoflurane 0.5-2%, using a positive end-expiratory pressure of 6 cm H2O. A CO2 pneumoperitoneum was established with a pressure-controlled high-flow insufflator; the intraabdominal pressure during the procedure was 14 mm Hg. Two hours after gas instillation, the peak airway pressure increased from 22 to 40 cm H2O, and the PaCO2 from 45 to 70 mm Hg. Breath sounds over the right lung were no longer heard, and subcutaneous emphysema was noted over the neck and face. An intraoperative chest X-ray confirmed a right pneumothorax. Following peritoneal gas evacuation, the PaCO2 returned to 35 mm Hg, the subcutaneous emphysema diminished, and a repeat chest X-ray showed complete resolution of the pneumothorax. The course of this event led us to the conclusion that the pneumothorax was due to diffusion of CO2 from the peritoneal to the pleural cavity through congenital defects in the diaphragm. Ureterolysis could be continued by laparotomy.
一位71岁的女性被诊断为继发于腹膜后纤维化的左侧输尿管狭窄。由于保守治疗可的松失败,腹腔镜输尿管溶解术。气管插管后,用40 vol% O2空气和0.5-2%异氟烷通气,呼气末正压为6 cm H2O。采用压力控制的高流量充气器建立CO2气腹;术中腹腔内压14 mm Hg,注气2 h后气道压力峰值由22 ~ 40 cm H2O升高,PaCO2由45 ~ 70 mm Hg升高,右肺无呼吸音,颈部及面部可见皮下肺气肿。术中胸部x光片证实右侧气胸。腹膜气体排出后,PaCO2恢复到35 mm Hg,皮下肺气肿减少,重复胸片显示气胸完全消退。这一事件的过程使我们得出结论,气胸是由于二氧化碳从腹膜扩散到胸膜腔通过先天性隔膜缺陷造成的。输尿管溶解可继续剖腹手术。