Diet intervention for modifying cancer risk.

D F Birt, J C Pelling, S Nair, D Lepley
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Abstract

Considerable evidence suggests that dietary differences between populations account for a significant proportion of the variation in cancer occurrence in different parts of the world. A major problem has been identifying the particular dietary components which predispose or protect individuals against cancer. For example, the high rates of breast and colon cancer in the United States have been associated with numerous dietary patterns including high fat, high dietary energy, and low fruit and vegetable intakes. Our laboratories have attempted to identify mechanisms whereby diet may modify cancer and it is anticipated that future studies will determine which of these potential mechanisms may be relevant in humans. A promising lead in understanding the mechanism of high dietary fat/high dietary energy promotion of cancer was the impact of these diets on cellular protein kinase C (PKC). PKC is important in cellular signaling events which are critical to tumor promotion. Our studies demonstrated increased PKC activity and/or protein expression observed in epidermis and pancreatic epithelial cells of rodents fed high fat/energy diets. The inverse association between cancer at a number of sites and fruit and vegetable intake may be due to both micronutrient and non-nutrient components of fruits and vegetables. We have studied the prevention of skin tumor promotion by apigenin, a plant flavonoid. Apigenin may block several points in the process of tumor promotion, including inhibiting kinases, reducing transcription factors and regulating cell cycle. The complexity of our diets and the multitude of potential dietary effects which may be important in cancer development make this a fertile area for future study.

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降低癌症风险的饮食干预。
大量证据表明,人口之间的饮食差异在世界不同地区癌症发病率的差异中占很大比例。一个主要的问题是确定哪些特定的饮食成分会使人易患癌症,哪些能保护人不患癌症。例如,在美国,乳腺癌和结肠癌的高发病率与多种饮食模式有关,包括高脂肪、高膳食能量和低水果和蔬菜摄入量。我们的实验室已经试图确定饮食可能改变癌症的机制,预计未来的研究将确定哪些潜在的机制可能与人类有关。高脂肪/高能量饮食对细胞蛋白激酶C (PKC)的影响是了解高脂肪/高能量饮食促进癌症的机制的一个有希望的线索。PKC在细胞信号事件中起重要作用,对肿瘤的促进至关重要。我们的研究表明,在喂食高脂肪/能量饮食的啮齿动物表皮和胰腺上皮细胞中观察到PKC活性和/或蛋白表达增加。许多部位的癌症与水果和蔬菜摄入量之间的负相关可能是由于水果和蔬菜中的微量营养素和非营养成分。我们研究了芹菜素(一种植物类黄酮)对皮肤肿瘤的预防作用。芹菜素可能阻断肿瘤促进过程中的几个点,包括抑制激酶、减少转录因子和调节细胞周期。我们饮食的复杂性和多种潜在的饮食影响可能对癌症的发展很重要,这使它成为未来研究的一个肥沃的领域。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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