Cerebellar-dependent learning as a neurobehavioral index of the cannabinoid system.

Chad R Edwards, Patrick D Skosnik
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引用次数: 17

Abstract

Delta-9-tetrahydrocannabinol (THC) is the primary psycho-active ingredient in Cannabis spp., the most widely used illicit drug in the United States. THC is an exogenous agonist of the central cannabinoid receptor (CB1), one of the most abundant G-coupled receptors in the mammalian brain. Although CB1 receptors are distributed throughout the brain, they are found at very high levels in the cerebellum. Despite the variety of disturbances associated with acute cannabis intoxication, including altered short-term memory, dissociation of thoughts, motor impairments, and paranoia, among others, a reliable index of cannabinoid system function has in large part eluded scientists. Thus, there is a demand in contemporary clinical neuroscience for methods sensitive to cannabinoid system function, not only for assessing how cannabis use influences human information processing, but also to assess the involvement of the endocannabinoid system (ECS) in clinical disease and evaluate the effects of CB1-based drug therapies. The purpose of the present article, therefore, is to address this current need by integrating two separate literatures. The first literature demonstrates that the ECS mediates synaptic plasticity, specifically, long-term depression (LTD) of parallel fibers at the parallel fiber-Purkinje junction in the cerebellar cortex. The second literature suggests that LTD at this junction is necessary for the acquisition of the primary dependent variable in delay eyeblink conditioning (EBC)--the exhibition of temporally measured conditioned responses. These two literatures are integrated by proposing an updated EBC circuit that incorporates the CB1 receptor and the endogenous cannabinoids. Finally, the implications of the model is discussed in consideration of recent evidence from CB1 knockout mice, human cannabis users, and schizophrenia patients, with the expectation that translational research on the cannabinoid system will be advanced.

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小脑依赖性学习作为大麻素系统的神经行为指标。
德尔塔-9-四氢大麻酚(THC)是大麻的主要精神活性成分,大麻是美国使用最广泛的非法药物。四氢大麻酚是中枢大麻素受体(CB1)的外源性激动剂,CB1是哺乳动物大脑中最丰富的g偶联受体之一。虽然CB1受体分布在整个大脑,但在小脑中发现的水平非常高。尽管与急性大麻中毒相关的各种紊乱,包括短期记忆改变、思想分离、运动障碍和偏执等,但在很大程度上,科学家们还没有找到大麻素系统功能的可靠指标。因此,当代临床神经科学需要对大麻素系统功能敏感的方法,不仅要评估大麻使用如何影响人类信息处理,还要评估内源性大麻素系统(ECS)在临床疾病中的作用,并评估基于cb1的药物治疗的效果。因此,本文的目的是通过整合两个独立的文献来解决当前的需求。第一个文献表明,ECS介导突触可塑性,特别是小脑皮层平行纤维-浦肯野交界处平行纤维的长期抑制(LTD)。第二篇文献表明,这个连接处的LTD对于延迟眨眼条件反射(EBC)的主要因变量的获取是必要的——时间测量条件反应的表现。这两篇文献通过提出一个更新的EBC电路整合了CB1受体和内源性大麻素。最后,考虑到CB1敲除小鼠、人类大麻使用者和精神分裂症患者的最新证据,讨论了该模型的意义,期望大麻素系统的转化研究将得到推进。
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