Attachment, Mentalization, and Criterion B of the Alternative DSM-5 Model for Personality Disorders (AMPD).

Ericka Ball Cooper, Jaime L Anderson, Carla Sharp, Hillary A Langley, Amanda Venta
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引用次数: 6

Abstract

Background: The mentalization theory posits that interpersonal difficulties and maladaptive personality traits develop from an insecure attachment pattern with one's caregiver and corresponding deficits in mentalizing-the ability to understand others' and one's own mental states. Mentalizing deficits have been theorized as the basis for all psychopathology, with the paradigmatic case being Borderline Personality Disorder. Nevertheless, developments in the personality field indicate personality pathology is best represented dimensionally, and such a proposal was outlined by the Alternative DSM-5 Model for Personality Disorders (AMPD). Despite evidence linking the mentalization theory to personality disorders, however, it has yet to be applied to Criterion B of the AMPD. The aim of the present study was to evaluate the moderating role of mentalizing in the relation between attachment and Criterion B maladaptive trait function in a sample of undergraduates. We hypothesized a model in which: (1) attachment insecurity would be positively associated with the Negative Affectivity, Antagonism, and Disinhibition personality domains; (2) mentalizing ability would be negatively associated with these domains; and, (3) there would be an interaction effect between attachment and mentalizing when predicting these same domains.

Methods: Personality domains were measured dimensionally via the Personality Inventory for DSM-5 (PID-5-SF), while the dependence and avoidance domains of attachment were assessed via the Relationship Questionnaire (RQ). Mentalizing ability was tapped by the Movie for the Assessment of Social Cognition (MASC). The AMPD personality domains and trait facets were examined as dependent variables; attachment dependence, attachment avoidance, and overall mentalizing ability were entered as independent variables; and interaction terms between mentalizing and each attachment dimension were used to test moderation via MANCOVAs.

Results: Consistent with expectations, results indicated overall mentalizing moderated the relation between attachment avoidance and Negative Affectivity. Posthoc analyses revealed similar effects on the relations between attachment avoidance and the Emotional Lability, Hostility, and Perseveration trait facets; however, there were no significant moderation findings related to attachment dependence.

Conclusions: These results support the mentalization theory's application to Criterion B of the AMPD, particularly in relation to the links between Negative Affectivity and borderline-related traits, and encourage future research of dimensional maladaptive personality. They further bolster support for understanding maladaptive personality as a dimensional construct.

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人格障碍(AMPD)的备选版DSM-5模型的依恋、心智化和标准B。
背景:心理化理论认为,人际交往困难和适应不良人格特征是由与照顾者的不安全依恋模式和相应的心理化缺陷(理解他人和自己心理状态的能力)发展而来的。心智缺陷已被理论化为所有精神病理学的基础,典型的例子是边缘型人格障碍。然而,人格领域的发展表明,人格病理学在维度上是最好的代表,这样的建议是由DSM-5人格障碍替代模型(AMPD)概述的。尽管有证据表明心智化理论与人格障碍有关,但它尚未适用于AMPD的B项标准。本研究旨在探讨心理化在大学生依恋与B标准适应不良特质功能关系中的调节作用。我们假设:(1)依恋不安全感与消极情感、对抗和去抑制人格域呈正相关;(2)心智化能力与这些领域呈负相关;(3)在预测这些领域时,依恋与心智化之间存在交互作用。方法:采用《精神疾病诊断与统计手册-5》(DSM-5 - sf)人格量表进行人格域测量,采用《关系问卷》(RQ)评估依恋依赖域和依恋回避域。心理化能力被电影用来评估社会认知。将AMPD人格域和特质方面作为因变量进行研究;依恋依赖、依恋回避和整体心智化能力作为自变量输入;心理化与依恋维度之间的相互作用项通过MANCOVAs来测试适度性。结果:与预期一致,整体心理化调节了依恋回避与消极情感的关系。事后分析显示,依恋回避与情绪不稳定、敌意和坚持特质方面的关系具有相似的影响;然而,没有显著的调节结果与依恋依赖有关。结论:这些结果支持了心理化理论在AMPD B标准中的应用,特别是在消极情感与边缘相关特征之间的联系方面,并鼓励了未来对维度适应不良人格的研究。他们进一步支持将适应不良人格理解为一种维度结构。
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来源期刊
CiteScore
6.00
自引率
9.80%
发文量
30
审稿时长
28 weeks
期刊介绍: Borderline Personality Disorder and Emotion Dysregulation provides a platform for researchers and clinicians interested in borderline personality disorder (BPD) as a currently highly challenging psychiatric disorder. Emotion dysregulation is at the core of BPD but also stands on its own as a major pathological component of the underlying neurobiology of various other psychiatric disorders. The journal focuses on the psychological, social and neurobiological aspects of emotion dysregulation as well as epidemiology, phenomenology, pathophysiology, treatment, neurobiology, genetics, and animal models of BPD.
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