Obesity and Atrial Fibrillation

J. T. Medeiros de Vasconcelos
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Abstract

Atrial fibrillation is currently a central issue in the universe of cardiac arrhythmias. The increase in life expectancy that brought a substantial increase in its prevalence in the general population1 implied a marked deepening in the understanding of its electrophysiological mechanisms, in the identification of determining factors or potentiation of its occurrence and obviously in the development of effective strategies of treatment and prevention of its complications. Since the classic study of Wijffels et al., published in 1995 (Atrial Fibrillation Begets Atrial Fibrillation)2 which experimentally demonstrated that atrial frequencies artificially imposed to the atrial myocardium imply marked electrophysiological changes determining the very occurrence of atrial fibrillation, it became clear that once arrhythmia is initiated it potentiates its own occurrence and a constant process of feedback, whose final outcome over time is the installation of arrhythmia in a permanent form. However, for atrial fibrillation to occur, the presence of an adequate electrophysiological environment is necessary, consequent to the presence of several elements that aggress the atrial myocardium under the electrical and structural aspects.
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肥胖与心房颤动
心房颤动目前是心律失常领域的一个核心问题。预期寿命的增加使其在普通人群中的患病率大幅增加1,这意味着对其电生理机制的理解、对其发生的决定因素或增强作用的识别以及对其并发症的有效治疗和预防策略的制定都有了显着的深化。自从Wijffels等人1995年发表的经典研究(心房颤动开始心房颤动)2实验证明人工施加在心房心肌上的心房频率意味着决定心房颤动发生的显著电生理变化以来,很明显,一旦心律失常开始,它会加剧自身的发生和持续的反馈过程,随着时间的推移,最终结果是心律失常以永久形式出现。然而,要想发生心房颤动,需要有足够的电生理环境,这是由于在电学和结构方面存在聚集心房心肌的几种元素。
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