COVID-19:通过mhc样诱饵系统攻击免疫细胞并干扰抗原呈递

IF 3.2 4区 医学 Q3 IMMUNOLOGY Journal of Immunotherapy Pub Date : 2023-04-01 DOI:10.1097/CJI.0000000000000455
Wenzhong Liu, Hualan Li
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引用次数: 1

摘要

2019冠状病毒病的高死亡率与抗原呈递不良和淋巴细胞减少有关。巨细胞病毒和疱疹家族编码一系列主要的组织相容性复合体(MHC)样分子,这些分子是靶向免疫反应实现免疫逃逸所必需的。在本研究中,结构域搜索结果显示,严重急性呼吸综合征冠状病毒2的许多蛋白具有mhc样结构域,类似于人类免疫系统的诱饵。MHC样结构可以与免疫细胞(如CD4 + t细胞、CD8 + t细胞和自然杀伤细胞)的MHC受体结合,干扰抗原呈递。当S蛋白的mhc样蛋白与免疫细胞结合后,E蛋白产生的氧自由基破坏免疫细胞。病毒蛋白(如S)的mhc样区发生突变,导致免疫抵抗力减弱,传播能力增强。S 127-194是delta型病毒强劲传播的主要原因。s144 -162调控S三聚体的形成。delta变异的RdRP: G671S和N: D63G突变导致病毒载量高。α、β、λ变异的S 62 ~ 80是快速传播的重要因子。s616 -676和1014-1114是伽马变异感染死亡率高的原因。这些位点位于mhc样结构区域。
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COVID-19: Attacks Immune Cells and Interferences With Antigen Presentation Through MHC-Like Decoy System.

The high mortality of coronavirus disease 2019 is related to poor antigen presentation and lymphopenia. Cytomegalovirus and the herpes family encode a series of major histocompatibility complex (MHC)-like molecules required for targeted immune responses to achieve immune escape. In this present study, domain search results showed that many proteins of the severe acute respiratory syndrome coronavirus 2 virus had MHC-like domains, which were similar to decoys for the human immune system. MHC-like structures could bind to MHC receptors of immune cells (such as CD4 + T-cell, CD8 + T-cell, and natural killer-cell), interfering with antigen presentation. Then the oxygen free radicals generated by E protein destroyed immune cells after MHC-like of S protein could bind to them. Mutations in the MHC-like region of the viral proteins such as S promoted weaker immune resistance and more robust transmission. S 127-194 were the primary reason for the robust transmission of delta variants. The S 144-162 regulated the formation of S trimer. The mutations of RdRP: G671S and N: D63G of delta variant caused high viral load. S 62-80 of alpha, beta, lambda variants were the important factor for fast-spreading. S 616-676 and 1014-1114 were causes of high mortality for gamma variants infections. These sites were in the MHC-like structure regions.

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来源期刊
Journal of Immunotherapy
Journal of Immunotherapy 医学-免疫学
CiteScore
6.90
自引率
0.00%
发文量
79
审稿时长
6-12 weeks
期刊介绍: Journal of Immunotherapy features rapid publication of articles on immunomodulators, lymphokines, antibodies, cells, and cell products in cancer biology and therapy. Laboratory and preclinical studies, as well as investigative clinical reports, are presented. The journal emphasizes basic mechanisms and methods for the rapid transfer of technology from the laboratory to the clinic. JIT contains full-length articles, review articles, and short communications.
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