磷化铝中毒死亡肺、肝组织病理学变化的应用

A. Mohammadi, S. Mohammadian, Maryam Zaare Nahandi
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引用次数: 0

摘要

在发展中国家,磷化铝是因接触杀虫剂而导致死亡的第三大原因。该物质与水或潮湿空气或酸接触产生磷化氢气体,通过身体各器官的细胞呼吸障碍导致细胞死亡(1)、(2)、(3)。近几十年来,磷化铝片很容易获得,并广泛用于伊朗人口的自杀企图(4),(5),(6),(7),(8),(9),(10),(11),(12)。磷化铝几乎累及人体所有重要器官,引起不同的临床特征(1)、(10),如中毒患者出现呼吸急促、发绀、双侧肺啰音、ALIARDS、肺水肿、肺不张、呼吸困难、呼吸衰竭、咳嗽、呼吸短促、胸闷(3)、(5)、(12)、胸腔积液(13)和急性呼吸骤停(14)等肺部症状。
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Application of histopathological changes in the lungs and liver of death from Aluminum Phosphide poisoning
In developing countries Aluminum Phosphide is the third cause of mortality due to contact with pesticides. This substance produces phosphine gas in contact with water or moist air or acid which causes cell death through cellular respiration disorder in various organs of the body (1),(2),(3). In recent decades Aluminum Phosphide tablets are easily available and widely used for suicidal attempts by Iranian population (4),(5),(6),(7),(8),(9),(10),(11),(12). Aluminum phosphide through involvement with almost all vital organs of the body causes different clinical features (1),(10) for example the poisoned patients catch pulmonary symptoms such as tachypnea, cyanosis, pulmonary rales bilaterally, ALIARDS, pulmonary edema, atelectasis, dyspnea, respiratory failure, cough, shortness of breath, tightness in chest (3),(5),(12), pleural effusion (13) and acute respiratory arrest (14).
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