{"title":"青蒿素配体对野生型恶性疟原虫Kelch13蛋白模型和C580Y、R539T、F446I突变型恶性疟原虫Kelch13蛋白模型的计算机相互作用研究","authors":"Semuel Sandy, Irawati Wike","doi":"10.1063/5.0104442","DOIUrl":null,"url":null,"abstract":"","PeriodicalId":383729,"journal":{"name":"10TH INTERNATIONAL CONFERENCE ON APPLIED SCIENCE AND TECHNOLOGY","volume":"96 1","pages":"0"},"PeriodicalIF":0.0000,"publicationDate":"1900-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Study in-silico interaction of artemisinin ligands against the wild-type Plasmodium falciparum Kelch13 protein model and the Plasmodium falciparum Kelch 13 protein model in mutations of C580Y, R539T, and F446I)\",\"authors\":\"Semuel Sandy, Irawati Wike\",\"doi\":\"10.1063/5.0104442\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"\",\"PeriodicalId\":383729,\"journal\":{\"name\":\"10TH INTERNATIONAL CONFERENCE ON APPLIED SCIENCE AND TECHNOLOGY\",\"volume\":\"96 1\",\"pages\":\"0\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"1900-01-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"10TH INTERNATIONAL CONFERENCE ON APPLIED SCIENCE AND TECHNOLOGY\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.1063/5.0104442\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"10TH INTERNATIONAL CONFERENCE ON APPLIED SCIENCE AND TECHNOLOGY","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.1063/5.0104442","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Study in-silico interaction of artemisinin ligands against the wild-type Plasmodium falciparum Kelch13 protein model and the Plasmodium falciparum Kelch 13 protein model in mutations of C580Y, R539T, and F446I)