感染性中毒性休克的发病机制及治疗。

Vrachebnoe delo Pub Date : 1991-11-01

L I Chuĭkov, S I Zablotskaia, I T Avdienko, A A Shvaĭchenko

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引用次数: 0

摘要

本文对56例感染性中毒性休克患者的血凝系统、血凝素和组胺进行了检测。严重休克的特点是高组胺血症，预钾likrein的过度激活以及钾likrein和组胺抑制剂的低代偿活性。凝血图的变化证明了pic综合征的表现。钾激肽-激肽系统的试验可作为纤溶血过程的指标。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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[The pathogenesis and treatment of infectious-toxic shock].

The kallikrein-kinin and coagulation systems of the blood and histamine were examined in 56 patients with infectious-toxic shock. Severe shock was characterized by high histaminemia, excessive activation of prekallikrein along with low compensatory activity of kallikrein and histamine inhibitors. Changes of the coagulograms evidence manifestations of the PIC-syndrome. Tests of the kallikrein-kinin system may serve as an index of fibrinolytic blood processes.

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