罕见的轻度自发性纵隔气肿伴哮喘加重1例。

Marco Umberto Scaramozzino, Sapone Giovanni, Levi Guido, Plastina Romeo Ubaldo
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引用次数: 0

摘要

导言:纵隔内存在自由空气,且与创伤无关,称为自发性纵隔气肿[1]。这种疾病最初是由Rene Laennec在1819年描述的。“自发”这个词是哈曼在1939年晚些时候引入的。这种情况可能是由于肺部受伤导致空气从纵隔渗漏而引起的。自发性气胸是指与创伤所致继发性气胸相比,发生在有肺部基础疾病或无任何医学相关疾病的患者中[2]。纵隔气肿的病理生理机制有6种:1)直接机制2)哈曼-麦克林机制3)筋膜下途径(继发于气胸或胸壁损伤)4)浆膜下途径:
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A Rare Case of Mild Spontaneous Pneumomediastinum Associated with Asthma Exacerbation.
Introduction: The presence of free air within the mediastinum, that is not associated with trauma, is known as spontaneous pneumomediastinum [1]. The disease was originally described by Rene Laennec in 1819. The word spontaneous was introduced by Hamman later in 1939.The condition can result from an injury that leads to air leakage into the mediastinum from the lung . Spontaneous indicates that it is encountered in patients with underlying lung diseases or without any medically relevant conditions as compared to that secondary pneumothorax that results from trauma [2]. The pathophysiological mechanisms underlying pneumomediastinum are six: 1) Direct mechanism 2) Hamman-Macklin mechanism 3) Subfascial route (secondary to pneumothorax or chest wall injury) 4) Subserosal route:
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