{"title":"双胞胎关系与儿童特应性疾病：了解不和双胞胎的早期生活因素","authors":"K. Zhang, J. Zhu, E. Terebessy, I. Fong, T. To","doi":"10.56164/pediatrrespirj.2023.34","DOIUrl":null,"url":null,"abstract":"Parents often question why one of their children would have asthma while their siblings do not. With twins, this is especially perplexing. The gene-environment interaction hypothesis suggests that early-life environmental exposures interact with genes, leading to asthma and atopic diseases. This study sought to investigate twins for potential early-life risk factors that modify the risk of asthma, allergic rhinitis","PeriodicalId":507533,"journal":{"name":"Pediatric Respiratory Journal","volume":null,"pages":null},"PeriodicalIF":0.0000,"publicationDate":"2023-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Twinship and childhood atopic disease: understanding early-life factors among discordant twins\",\"authors\":\"K. Zhang, J. Zhu, E. Terebessy, I. Fong, T. To\",\"doi\":\"10.56164/pediatrrespirj.2023.34\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Parents often question why one of their children would have asthma while their siblings do not. With twins, this is especially perplexing. The gene-environment interaction hypothesis suggests that early-life environmental exposures interact with genes, leading to asthma and atopic diseases. This study sought to investigate twins for potential early-life risk factors that modify the risk of asthma, allergic rhinitis\",\"PeriodicalId\":507533,\"journal\":{\"name\":\"Pediatric Respiratory Journal\",\"volume\":null,\"pages\":null},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2023-11-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Pediatric Respiratory Journal\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.56164/pediatrrespirj.2023.34\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Pediatric Respiratory Journal","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.56164/pediatrrespirj.2023.34","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Twinship and childhood atopic disease: understanding early-life factors among discordant twins
Parents often question why one of their children would have asthma while their siblings do not. With twins, this is especially perplexing. The gene-environment interaction hypothesis suggests that early-life environmental exposures interact with genes, leading to asthma and atopic diseases. This study sought to investigate twins for potential early-life risk factors that modify the risk of asthma, allergic rhinitis
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