镉毒性与自噬:综述。

IF 4.1 3区 生物学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY Biometals Pub Date : 2024-06-01 Epub Date: 2024-01-26 DOI:10.1007/s10534-023-00581-y
Yueting Shao, Liting Zheng, Yiguo Jiang
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引用次数: 0

摘要

镉(Cd)是一种对人类健康构成威胁的重要环境污染物,是空气污染物、食物来源和香烟烟雾的重要组成部分。镉是一种已知的致癌物质,对环境和人体各器官都有毒性影响。生物体内的重金属难以生物降解,进入呼吸道的重金属也难以清除。自噬是抵御细胞外(微生物和异物)或细胞内(蛋白酶体无法降解的受损细胞器和蛋白质)压力的一种关键机制,是真核生物抵御重金属毒性的一种自我保护机制。自噬通过隔离和收集与其他分子事件相关的外来化学物质信息来维持细胞的平衡。然而,在某些病理条件下,包括癌症,自噬可能会引发细胞死亡。自噬功能障碍是镉诱导细胞毒性的主要机制之一。本综述评估了镉诱导的自噬对不同人体器官系统的毒性影响,重点关注肝毒性、肾毒性、呼吸系统毒性和神经毒性。这篇综述还强调了镉诱导自噬的经典分子途径,包括 ROS 依赖性信号途径、内质网(ER)应激途径、哺乳动物雷帕霉素靶标(mTOR)途径、Beclin-1 和 Bcl-2 家族,以及最近发现的与镉相关的分子。此外,还提出了有关自噬功能的镉毒性研究方向。本综述介绍了全面揭示自噬在镉毒性反应中行为的最新理论,并提出了针对镉毒性和人类镉相关疾病的潜在自噬预防和治疗策略。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

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Cadmium toxicity and autophagy: a review.

Cadmium (Cd) is an important environmental pollutant that poses a threat to human health and represents a critical component of air pollutants, food sources, and cigarette smoke. Cd is a known carcinogen and has toxic effects on the environment and various organs in humans. Heavy metals within an organism are difficult to biodegrade, and those that enter the respiratory tract are difficult to remove. Autophagy is a key mechanism for counteracting extracellular (microorganisms and foreign bodies) or intracellular (damaged organelles and proteins that cannot be degraded by the proteasome) stress and represents a self-protective mechanism for eukaryotes against heavy metal toxicity. Autophagy maintains cellular homeostasis by isolating and gathering information about foreign chemicals associated with other molecular events. However, autophagy may trigger cell death under certain pathological conditions, including cancer. Autophagy dysfunction is one of the main mechanisms underlying Cd-induced cytotoxicity. In this review, the toxic effects of Cd-induced autophagy on different human organ systems were evaluated, with a focus on hepatotoxicity, nephrotoxicity, respiratory toxicity, and neurotoxicity. This review also highlighted the classical molecular pathways of Cd-induced autophagy, including the ROS-dependent signaling pathways, endoplasmic reticulum (ER) stress pathway, Mammalian target of rapamycin (mTOR) pathway, Beclin-1 and Bcl-2 family, and recently identified molecules associated with Cd. Moreover, research directions for Cd toxicity regarding autophagic function were proposed. This review presents the latest theories to comprehensively reveal autophagy behavior in response to Cd toxicity and proposes novel potential autophagy-targeted prevention and treatment strategies for Cd toxicity and Cd-associated diseases in humans.

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来源期刊
Biometals
Biometals 生物-生化与分子生物学
CiteScore
5.90
自引率
8.60%
发文量
111
审稿时长
3 months
期刊介绍: BioMetals is the only established journal to feature the important role of metal ions in chemistry, biology, biochemistry, environmental science, and medicine. BioMetals is an international, multidisciplinary journal singularly devoted to the rapid publication of the fundamental advances of both basic and applied research in this field. BioMetals offers a forum for innovative research and clinical results on the structure and function of: - metal ions - metal chelates, - siderophores, - metal-containing proteins - biominerals in all biosystems. - BioMetals rapidly publishes original articles and reviews. BioMetals is a journal for metals researchers who practice in medicine, biochemistry, pharmacology, toxicology, microbiology, cell biology, chemistry, and plant physiology who are based academic, industrial and government laboratories.
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