撤稿:"MiR-137通过靶向MRGBP在胰腺癌中发挥抑癌作用

IF 3 3区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY Journal of cellular biochemistry Pub Date : 2024-04-01 DOI:10.1002/jcb.30552
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引用次数: 0

摘要

撤稿:"丁锋、张爽、高少阳、尚健、李艳霞、崔宁、赵秋:《MiR-137通过靶向MRGBP发挥胰腺癌肿瘤抑制因子的功能》,《细胞生物化学杂志》(J Cell Biochem.2018;4799-4807:上述文章于2018年1月13日在线发表于《Wiley Online Library》(https://doi.org/10.1002/jcb.26676),经该刊主编Christian Behl教授博士与Wiley Periodicals LLC达成协议,该文章已被撤回。作者表示,在图表编译过程中出现了错误,文章中的实验数据无法核实,因此同意撤稿。此外,调查还发现一些图片元素存在不一致之处。因此,编辑认为这篇文章的结论无效。
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Retraction: “MiR-137 functions as a tumor suppressor in pancreatic cancer by targeting MRGBP”

Retraction: “MiR-137 functions as a tumor suppressor in pancreatic cancer by targeting MRGBP” by Feng Ding, Shuang Zhang, Shaoyang Gao, Jian Shang, Yanxia Li, Ning Cui, and Qiu Zhao, J Cell Biochem. 2018; 4799-4807: The above article, published online on 13 January 2018 in Wiley Online Library (https://doi.org/10.1002/jcb.26676) has been retracted by agreement between the journal's Editor in Chief, Prof. Dr. Christian Behl, and Wiley Periodicals LLC.

The retraction has been agreed after the authors stated that errors occurred during figure compilation, and that the experimental data in the article could not be verified. The investigation additionally revealed inconsistencies in several image elements. Thus, the editors consider the conclusions of this article to be invalid.

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来源期刊
Journal of cellular biochemistry
Journal of cellular biochemistry 生物-生化与分子生物学
CiteScore
9.90
自引率
0.00%
发文量
164
审稿时长
1 months
期刊介绍: The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.
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