撤稿:"长非编码RNA SNHG1的下调通过抑制胰腺癌的Notch-1信号通路,抑制细胞增殖、转移和侵袭"

IF 3 3区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY Journal of cellular biochemistry Pub Date : 2024-04-09 DOI:10.1002/jcb.30550
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引用次数: 0

摘要

撤稿:"Downregulation of long noncoding RNA SNHG1 inhibits cell proliferation, metastasis, and invasion by suppressing the Notch-1 signaling pathway in pancreatic cancer" by Long Cui, Yadong Dong, Xiaochuan Wang, Xin Zhao, Chenchen Kong, Yangsui Liu, Xinchun Jiang, Xinhui Zhang, J Cell Biochem 2019, 120: 6106-6112.上述文章于2018年12月5日在线发表于《Wiley Online Library》(https://onlinelibrary.wiley.com/doi/10.1002/jcb.27897),经作者、该杂志主编Christian Behl和Wiley Periodicals LLC三方协商,已同意撤回该文章。由于对文章中数据的担忧,经作者请求,已同意撤回该文章。作者承认在图表编辑过程中出现了一些错误,导致所提供的结果与所描述的实验方法之间存在缺陷和不一致。因此,这篇文章的结论被认为是无效的。
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Retraction: “Downregulation of long noncoding RNA SNHG1 inhibits cell proliferation, metastasis, and invasion by suppressing the Notch-1 signaling pathway in pancreatic cancer”

Retraction: “Downregulation of long noncoding RNA SNHG1 inhibits cell proliferation, metastasis, and invasion by suppressing the Notch-1 signaling pathway in pancreatic cancer” by Long Cui, Yadong Dong, Xiaochuan Wang, Xin Zhao, Chenchen Kong, Yangsui Liu, Xinchun Jiang, Xinhui Zhang, J Cell Biochem 2019, 120: 6106-6112. The above article, published online on 5 December 2018 in Wiley Online Library (https://onlinelibrary.wiley.com/doi/10.1002/jcb.27897) has been retracted by agreement between the authors, the journal's Editor in Chief, Christian Behl, and Wiley Periodicals LLC.

The retraction has been agreed upon authors’ request due to concerns related to the data presented in the article. The authors admitted several mistakes during figure compilation resulting in flaws and inconsistencies between results presented and experimental methods described. Thus, the conclusions of this article are considered invalid.

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来源期刊
Journal of cellular biochemistry
Journal of cellular biochemistry 生物-生化与分子生物学
CiteScore
9.90
自引率
0.00%
发文量
164
审稿时长
1 months
期刊介绍: The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.
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