LPCAT1 介导的膜磷脂重塑促进了铁变态反应的逃避和肿瘤的生长

IF 17.3 1区 生物学 Q1 CELL BIOLOGY Nature Cell Biology Pub Date : 2024-04-26 DOI:10.1038/s41556-024-01405-y
Ziwen Li, Yameng Hu, Haiqing Zheng, Man Li, Yuanji Liu, Rongni Feng, Xincheng Li, Shuxia Zhang, Miaoling Tang, Meisongzhu Yang, Ruyuan Yu, Yingru Xu, Xinyi Liao, Suwen Chen, Wanying Qian, Qiliang Zhang, Daolin Tang, Bo Li, Libing Song, Jun Li
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摘要

细胞膜磷脂的动态重塑是为了防止磷脂过氧化引起的膜损伤并逃避铁中毒(一种由铁依赖的脂质过氧化驱动的非凋亡性细胞死亡形式),但人们对其背后的机制仍然知之甚少。在这里,我们发现溶血磷脂酰胆碱酰基转移酶1(LPCAT1)通过Lands循环提高膜磷脂饱和度,从而降低膜多不饱和脂肪酸水平,保护细胞免受磷脂过氧化诱导的膜损伤并抑制铁卟啉中毒,从而在抗铁卟啉中毒过程中发挥关键作用。此外,肿瘤细胞体内肿瘤形成能力的增强与 LPCAT1 的上调和铁变态反应抗性状态的出现密切相关。将 LPCAT1 抑制与铁凋亡诱导剂结合可协同触发铁凋亡并抑制肿瘤生长。因此,我们的研究结果揭示了 LPCAT1 在逃避铁变态反应中的合理作用,并建议将其作为人类癌症临床干预的一个有前途的靶点。
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LPCAT1-mediated membrane phospholipid remodelling promotes ferroptosis evasion and tumour growth
The mechanisms underlying the dynamic remodelling of cellular membrane phospholipids to prevent phospholipid peroxidation-induced membrane damage and evade ferroptosis, a non-apoptotic form of cell death driven by iron-dependent lipid peroxidation, remain poorly understood. Here we show that lysophosphatidylcholine acyltransferase 1 (LPCAT1) plays a critical role in ferroptosis resistance by increasing membrane phospholipid saturation via the Lands cycle, thereby reducing membrane levels of polyunsaturated fatty acids, protecting cells from phospholipid peroxidation-induced membrane damage and inhibiting ferroptosis. Furthermore, the enhanced in vivo tumour-forming capability of tumour cells is closely associated with the upregulation of LPCAT1 and emergence of a ferroptosis-resistant state. Combining LPCAT1 inhibition with a ferroptosis inducer synergistically triggers ferroptosis and suppresses tumour growth. Therefore, our results unveil a plausible role for LPCAT1 in evading ferroptosis and suggest it as a promising target for clinical intervention in human cancer.
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来源期刊
Nature Cell Biology
Nature Cell Biology 生物-细胞生物学
CiteScore
28.40
自引率
0.90%
发文量
219
审稿时长
3 months
期刊介绍: Nature Cell Biology, a prestigious journal, upholds a commitment to publishing papers of the highest quality across all areas of cell biology, with a particular focus on elucidating mechanisms underlying fundamental cell biological processes. The journal's broad scope encompasses various areas of interest, including but not limited to: -Autophagy -Cancer biology -Cell adhesion and migration -Cell cycle and growth -Cell death -Chromatin and epigenetics -Cytoskeletal dynamics -Developmental biology -DNA replication and repair -Mechanisms of human disease -Mechanobiology -Membrane traffic and dynamics -Metabolism -Nuclear organization and dynamics -Organelle biology -Proteolysis and quality control -RNA biology -Signal transduction -Stem cell biology
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