返回:"冈田酸通过抑制蛋白磷酸酶 2A 促进肝癌细胞的上皮-间质转化"。

IF 3 3区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY Journal of cellular biochemistry Pub Date : 2024-05-22 DOI:10.1002/jcb.30607
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引用次数: 0

摘要

撤回:J. Kong, D. Li, S. Zhang, H. Zhang, Y. Fu, B. Qian, C. Bei, S. Tan, X. Zhu, "Okadaic acid promotes epithelial-mesenchymal transition of hepatocellular carcinoma cells by inhibiting protein phosphatase 2A," Journal of Cellular Biochemistry 122, no:993-1002,https://doi.org/10.1002/jcb.29629 上述文章于 2020 年 1 月 6 日在线发表于 Wiley Online Library (wileyonlinelibrary.com),经作者、期刊主编 Christian Behl 和 Wiley Periodicals LLC 协议撤回。撤稿协议是根据第三方提出的指控进行调查后达成的。在调查过程中,发现所提供的数据存在若干缺陷和不一致之处。作者承认在本稿件的图表编辑过程中出现了重大失误。因此,编辑认为这篇文章的结论无效。
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RETRACTION: “Okadaic acid promotes epithelial-mesenchymal transition of hepatocellular carcinoma cells by inhibiting protein phosphatase 2A”

RETRACTION: J. Kong, D. Li, S. Zhang, H. Zhang, Y. Fu, B. Qian, C. Bei, S. Tan, X. Zhu, “Okadaic acid promotes epithelial-mesenchymal transition of hepatocellular carcinoma cells by inhibiting protein phosphatase 2A,” Journal of Cellular Biochemistry 122, no. 9 (2021): 993-1002, https://doi.org/10.1002/jcb.29629

The above article, published online on 6 January 2020 in Wiley Online Library (wileyonlinelibrary.com) has been retracted by agreement between the authors; the journal's Editor in Chief, Christian Behl; and Wiley Periodicals LLC.

The retraction has been agreed following an investigation based on allegations raised by third parties. During the investigation, several flaws and inconsistencies were found within the provided data. The authors admitted substantial mistakes during figure compilation for this manuscript. Thus, the conclusions of this article are considered invalid by the editors.

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来源期刊
Journal of cellular biochemistry
Journal of cellular biochemistry 生物-生化与分子生物学
CiteScore
9.90
自引率
0.00%
发文量
164
审稿时长
1 months
期刊介绍: The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.
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