封面图片，第 72 卷第 8 期

IF 5.1 2区医学 Q1 NEUROSCIENCES Glia Pub Date : 2024-06-28 DOI:10.1002/glia.24407

Sarah Wirth, Annika Schlößer, Antonia Beiersdorfer, Michaela Schweizer, Marcel S. Woo, Manuel A. Friese, Christian Lohr, Katarzyna M. Grochowska

{"title":"封面图片，第 72 卷第 8 期","authors":"Sarah Wirth, Annika Schlößer, Antonia Beiersdorfer, Michaela Schweizer, Marcel S. Woo, Manuel A. Friese, Christian Lohr, Katarzyna M. Grochowska","doi":"10.1002/glia.24407","DOIUrl":null,"url":null,"abstract":"<p>Cover Illustration: The upregulation of glial fibrillary acidic protein (GFAP; fire lookup table, the brightness of the color is proportional to the pixel intensity), an intermediate filament protein, is a marker of astrocytic reactivity associated with Alzheimer's disease (AD) pathology. The treatment of cultured astrocytes with a modified form of amyloid-β, Aβ3(pE)- 42, leads to the prominent GFAP upregulation. (See Wirth, S., et al, https://doi.org/10.1002/glia.24539)\n\n <figure>\n <div><picture>\n <source></source></picture><p></p>\n </div>\n </figure>\n </p>","PeriodicalId":174,"journal":{"name":"Glia","volume":"72 8","pages":"C1"},"PeriodicalIF":5.1000,"publicationDate":"2024-06-28","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1002/glia.24407","citationCount":"0","resultStr":"{\"title\":\"Cover Image, Volume 72, Issue 8\",\"authors\":\"Sarah Wirth, Annika Schlößer, Antonia Beiersdorfer, Michaela Schweizer, Marcel S. Woo, Manuel A. Friese, Christian Lohr, Katarzyna M. Grochowska\",\"doi\":\"10.1002/glia.24407\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>Cover Illustration: The upregulation of glial fibrillary acidic protein (GFAP; fire lookup table, the brightness of the color is proportional to the pixel intensity), an intermediate filament protein, is a marker of astrocytic reactivity associated with Alzheimer's disease (AD) pathology. The treatment of cultured astrocytes with a modified form of amyloid-β, Aβ3(pE)- 42, leads to the prominent GFAP upregulation. (See Wirth, S., et al, https://doi.org/10.1002/glia.24539)\\n\\n <figure>\\n <div><picture>\\n <source></source></picture><p></p>\\n </div>\\n </figure>\\n </p>\",\"PeriodicalId\":174,\"journal\":{\"name\":\"Glia\",\"volume\":\"72 8\",\"pages\":\"C1\"},\"PeriodicalIF\":5.1000,\"publicationDate\":\"2024-06-28\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1002/glia.24407\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Glia\",\"FirstCategoryId\":\"3\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1002/glia.24407\",\"RegionNum\":2,\"RegionCategory\":\"医学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q1\",\"JCRName\":\"NEUROSCIENCES\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Glia","FirstCategoryId":"3","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1002/glia.24407","RegionNum":2,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q1","JCRName":"NEUROSCIENCES","Score":null,"Total":0}

引用次数: 0

摘要

封面插图：神经胶质纤维酸性蛋白（GFAP；火烧查找表，颜色亮度与像素强度成正比）是一种中间丝蛋白，它的上调是与阿尔茨海默病（AD）病理相关的星形胶质细胞反应性的标志。用一种改良形式的淀粉样蛋白-β（Aβ3(pE)- 42）处理培养的星形胶质细胞会导致突出的 GFAP 上调。(见 Wirth, S. 等人，https://doi.org/10.1002/glia.24539）

本文章由计算机程序翻译，如有差异，请以英文原文为准。

摘要图片

查看原文

微信好友朋友圈 QQ好友复制链接

本刊更多论文

Cover Image, Volume 72, Issue 8

Cover Illustration: The upregulation of glial fibrillary acidic protein (GFAP; fire lookup table, the brightness of the color is proportional to the pixel intensity), an intermediate filament protein, is a marker of astrocytic reactivity associated with Alzheimer's disease (AD) pathology. The treatment of cultured astrocytes with a modified form of amyloid-β, Aβ3(pE)- 42, leads to the prominent GFAP upregulation. (See Wirth, S., et al, https://doi.org/10.1002/glia.24539)

求助全文

通过发布文献求助，成功后即可免费获取论文全文。去求助

来源期刊

Glia 医学-神经科学

CiteScore

13.10

自引率

4.80%

发文量

162

审稿时长

3-8 weeks

期刊介绍： GLIA is a peer-reviewed journal, which publishes articles dealing with all aspects of glial structure and function. This includes all aspects of glial cell biology in health and disease.