LAIR1 通过激活 AKT-IKKβ-p65 轴促进肝癌细胞转移并诱导 M2-巨噬细胞浸润。

IF 3 2区 医学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY Molecular Carcinogenesis Pub Date : 2024-09-01 Epub Date: 2024-07-17 DOI:10.1002/mc.23776
Banglun Pan, Shuling Shen, Jun Zhao, Zhu Zhang, Dongjie Ye, Xiaoxia Zhang, Yuxin Yao, Yue Luo, Xiaoqian Wang, Nanhong Tang
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引用次数: 0

摘要

LAIR1是免疫细胞上的一种受体,能够与胶原蛋白结合,并参与免疫相关疾病的发生。然而,肝细胞癌(HCC)细胞上表达的 LAIR1 对肿瘤微环境的确切贡献仍不清楚。我们的研究采用生物信息学分析和免疫荧光技术研究 LAIR1 水平与临床指标之间的相关性。我们采用了Transwell和划痕试验来评估LAIR1如何影响HCC细胞的迁移和侵袭。利用 RT-qPCR、transwell 和免疫荧光研究了巨噬细胞的趋化能力和替代性活化。为了研究其分子机制,我们采用了转录组测序分析、Western 印迹、细胞核/细胞质分馏、ELISA 和细胞因子芯片。我们发现 LAIR1 的存在与 HCC 的不良预后之间存在明显的相关性。我们发现 LAIR1 通过 AKT-IKKβ-p65 轴促进了 HCC 细胞的迁移和侵袭。此外,LAIR1 诱导的肿瘤相关巨噬细胞的替代性活化和浸润分别依赖于该轴中 IL6 和 CCL5 的上调。总之,研究发现阻断 LAIR1 是对抗 HCC 癌症发展的有效方法。
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LAIR1 promotes hepatocellular carcinoma cell metastasis and induces M2-macrophage infiltration through activating AKT-IKKβ-p65 axis.

LAIR1, a receptor found on immune cells, is capable of binding to collagen and is involved in immune-related diseases. However, the precise contribution of LAIR1 expressed on hepatocellular carcinoma (HCC) cells to tumor microenvironment is still unclear. In our study, bioinformatics analysis and immunofluorescence were employed to study the correlation between LAIR1 levels and clinical indicators. Transwell and scratch tests were used to evaluate how LAIR1 affected the migration and invasion of HCC cells. The chemotactic capacity and alternative activation of macrophages were investigated using RT-qPCR, transwell, and immunofluorescence. To investigate the molecular mechanisms, transcriptome sequencing analysis, Western blot, nucleus/cytoplasm fractionation, ELISA, and cytokine microarray were employed. We revealed a significant correlation between the presence of LAIR1 and an unfavorable outcome in HCC. We indicated that LAIR1 promoted migration and invasion of HCC cells through the AKT-IKKβ-p65 axis. Additionally, the alternative activation and infiltration of tumor-associated macrophages induced by LAIR1 were reliant on the upregulation of IL6 and CCL5 within this axis, respectively. In conclusion, blocking LAIR1 was found to be an effective approach in combating the cancerous advancement of HCC.

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来源期刊
Molecular Carcinogenesis
Molecular Carcinogenesis 医学-生化与分子生物学
CiteScore
7.30
自引率
2.20%
发文量
112
审稿时长
2 months
期刊介绍: Molecular Carcinogenesis publishes articles describing discoveries in basic and clinical science of the mechanisms involved in chemical-, environmental-, physical (e.g., radiation, trauma)-, infection and inflammation-associated cancer development, basic mechanisms of cancer prevention and therapy, the function of oncogenes and tumors suppressors, and the role of biomarkers for cancer risk prediction, molecular diagnosis and prognosis.
期刊最新文献
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