阿育吠陀在治疗贝克肌肉萎缩症中的作用:病例研究

Mohita Bohra
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摘要

肌肉萎缩症是一组遗传性疾病,会导致肌肉逐渐变弱,随着时间的推移,残疾程度会增加。贝克尔肌肉营养不良症(BMD)就是这样一种疾病,它是通过 X 连锁隐性模式遗传的,主要影响男性。贝克尔肌肉萎缩症的症状通常在十几岁或二十几岁时开始出现。最初的症状可能包括运动时肌肉痉挛和耐力下降。随着时间的推移,臀部、骨盆、大腿和肩部的肌肉会逐渐松弛,通常会导致用脚趾走路的倾向。阿育吠陀认为,导致这种疾病的因素是瓦塔(三体之一)的加重、Mamsa(肌肉组织)和 Meda(脂肪组织)的消耗、消化系统火力受损以及功能性通道(Srotorodha)的堵塞。本病例中的患者是一名 15 岁的男性,双下肢无力已有 4 年,骨盆和大腿周围的肌肉量减少已有 3 年,小腿肌肉体积增大已有 3 年。患者双小腿肌肉呈假性肥大,高尔氏征阳性,家族史阳性,肌酸磷酸激酶水平升高,因此诊断为 BMD。治疗方法结合了潘查卡玛疗法和阿育吠陀内服配方,以治疗 Sarvang Vata 失调症。治疗分四个疗程进行,每个疗程之间间隔 3 个月。经过一年的治疗,患者的病情不仅停止了发展,而且下肢力量也有了明显改善,从 4 增强到了 4+。此外,近端肌肉质量也有所增加,从 12.5 英寸增加到 13 英寸,小腿肌肉体积和紧绷感也有所减轻。这一令人鼓舞的结果表明,利用阿育吠陀原则和疗法来控制疾病是有潜力的。
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Role of Ayurveda in the management of becker muscular dystrophy: A case study
Muscular dystrophies are a group of genetic disorders that lead to a gradual weakening of muscles, resulting in increased disability over time. Becker muscular dystrophy (BMD) is one such condition and is inherited through an X-linked recessive pattern, primarily affecting males. Symptoms of BMD typically begin in the teenage years or late twenties. Initial signs may include muscle cramps during exercise and reduced stamina. Over time, there is a gradual loss of muscle bulk in the hips, pelvis, thighs, and shoulders, often leading to a tendency to walk on toes. In Ayurveda, the factors contributing to this disease are believed to be an aggravation of Vata (one of the three doshas), depletion of Mamsa (muscle tissue), and Meda (fat tissue), impaired digestive fire, and blockage of functional channels (Srotorodha). The present case involved a 15-year-old male with weakness in both lower limbs for 4 years, a decrease in muscle mass around the pelvic and thigh area for 3 years, and an increase in calf muscle size for 3 years. The patient exhibited pseudohypertrophy in both calf muscles and positive Gower's sign, along with a positive family history and elevated creatine phosphokinase levels, leading to a diagnosis of BMD. The treatment approach incorporated Panchakarma therapies and internal Ayurvedic formulations in line with the management of Sarvang Vata disorder. The treatment was administered in four sessions, with a 3-month gap between each therapy session. After a year of treatment, there was not only a halt in the progression of the disease but also a notable improvement in lower limb strength, which increased from 4 to 4+. In addition, there was an increase in proximal muscle mass, from 12.5 inches to 13 inches, and a reduction in calf muscle size and tightness. This promising outcome suggests the potential for managing the disease using Ayurvedic principles and therapies.
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