鉴定 APPB1 作为无性淋巴瘤激酶的底物。

IF 2.1 4区 生物学 Q4 BIOCHEMISTRY & MOLECULAR BIOLOGY Journal of biochemistry Pub Date : 2024-11-04 DOI:10.1093/jb/mvae055
Yuji Suzuki, Shoma Tsubota, Kenji Kadomatsu, Kazuma Sakamoto
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引用次数: 0

摘要

无性淋巴瘤激酶(ALK)是一种众所周知的致癌基因,与多种恶性肿瘤有关,如无性大细胞淋巴瘤、肺癌和神经母细胞瘤。融合 ALK 的几种底物已被确定,其生物学功能也已被描述。然而,由于缺乏对ALK底物的全面鉴定,限制了我们对受体ALK生物学作用的了解。因此,本研究旨在鉴定新型 ALK 底物并描述其生物学功能。我们利用亲近依赖性生物素鉴定筛选了受体 ALK 激酶结构域的相互作用者,并鉴定出 43 个相互作用者。我们在神经母细胞瘤细胞系 NB-1 中评估了这些相互作用物是否是 ALK 的下游,从而缩小了候选相互作用物的范围。其中,我们发现淀粉样 beta 前体蛋白结合家族 B 成员 1(APBB1)是参与 NB-1 细胞活力的 ALK 下游分子。最后,我们评估了ALK和APBB1之间的激酶-底物关系,发现ALK在细胞内和试管内实验中均使APBB1的多个酪氨酸残基磷酸化,其中酪氨酸269是一个主要靶点。总之,我们成功鉴定了受体 ALK 的一种新底物。我们的研究结果可能有助于进一步阐明神经母细胞瘤中ALK下游信号转导的分子机制。
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Identification of APBB1 as a substrate for anaplastic lymphoma kinase.

Anaplastic lymphoma kinase (ALK) is a well-known oncogene involved in various malignancies such as anaplastic large cell lymphoma, lung cancer and neuroblastoma. Several substrates for fused ALK have been identified and their biological functions have been described. However, the lack of a comprehensive identification of ALK substrates limits our understanding of the biological roles of receptor ALK. Thus, this study aimed to identify novel ALK substrates and characterize their biological functions. We screened the interactors of the kinase domain of receptor ALK using proximity-dependent biotin identification and identified 43 interactors. We narrowed down the candidates by evaluating whether these interactors were downstream of ALK in a neuroblastoma cell line, NB-1. Amongst these, we identified amyloid beta precursor protein-binding family B member 1 (APBB1) as an ALK downstream molecule involved in NB-1 cell viability. Finally, we assessed the kinase-substrate relationship between ALK and APBB1 and found that ALK phosphorylated multiple tyrosine residues in APBB1 both in-cell and in-tube assays, with tyrosine 269 as a major target. In conclusion, we successfully identified a new substrate for receptor ALK. Our results may help further elucidate the molecular mechanism of ALK downstream signalling in neuroblastoma.

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来源期刊
Journal of biochemistry
Journal of biochemistry 生物-生化与分子生物学
CiteScore
4.80
自引率
3.70%
发文量
101
审稿时长
4-8 weeks
期刊介绍: The Journal of Biochemistry founded in 1922 publishes the results of original research in the fields of Biochemistry, Molecular Biology, Cell, and Biotechnology written in English in the form of Regular Papers or Rapid Communications. A Rapid Communication is not a preliminary note, but it is, though brief, a complete and final publication. The materials described in Rapid Communications should not be included in a later paper. The Journal also publishes short reviews (JB Review) and papers solicited by the Editorial Board.
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