对 "通过抑制 NLRP3 炎症小体激活 NR1H3 减轻脓毒症心肌损伤的严重程度 "的更正

IF 6.1 2区 医学 Q1 ENGINEERING, BIOMEDICAL Bioengineering & Translational Medicine Pub Date : 2024-08-15 DOI:10.1002/btm2.10707
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引用次数: 0

摘要

邓超、刘琼、赵华东、钱璐、雷万瑞、杨雯雯、梁振兴、田野、张少飞、王长宇、陈颖、杨洋 通过抑制NLRP3炎性体激活NR1H3减轻脓毒症心肌损伤的严重程度Bioeng Transl Med. 2023;8(3):e10517.在已发表文章的图 2c 中发现 ABCA1 的统计图表有误。我们对这一错误表示歉意。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

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Correction to “Activation of NR1H3 attenuates the severity of septic myocardial injury by inhibiting NLRP3 inflammasome”

Chao Deng, Qiong Liu, Huadong Zhao, Lu Qian, Wagnrui Lei, Wenwen Yang, Zhenxing Liang, Ye Tian, Shaofei Zhang, Changyu Wang, Ying Chen, Yang Yang. Activation of NR1H3 attenuates the severity of septic myocardial injury by inhibiting NLRP3 inflammasome. Bioeng Transl Med. 2023; 8(3):e10517.

An inaccuracy has been found in the statistical graph of ABCA1 in Figure 2c of the published article. The corrected version of Figure 2 is shown below.

We apologize for this error.

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来源期刊
Bioengineering & Translational Medicine
Bioengineering & Translational Medicine Pharmacology, Toxicology and Pharmaceutics-Pharmaceutical Science
CiteScore
8.40
自引率
4.10%
发文量
150
审稿时长
12 weeks
期刊介绍: Bioengineering & Translational Medicine, an official, peer-reviewed online open-access journal of the American Institute of Chemical Engineers (AIChE) and the Society for Biological Engineering (SBE), focuses on how chemical and biological engineering approaches drive innovative technologies and solutions that impact clinical practice and commercial healthcare products.
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