鉴定 PI4K2A 中的 GABARAP 结合决定因子。

IF 3.8 3区 生物学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY Bioscience Reports Pub Date : 2024-10-30 DOI:10.1042/BSR20240200
Yan Chen, Barbara Barylko, John P Eichorst, Joachim D Mueller, Joseph P Albanesi
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引用次数: 0

摘要

GABARAP 是泛素样自噬相关蛋白 ATG8 家族的成员。它最初是作为 GABA-A 受体向质膜转运的促进因子被发现的,后来又被证明能在非自噬条件下促进多种其他蛋白质的胞内转运。我们和其他人已经证明,GABARAP 与 II 型磷脂酰肌醇 4- 激酶 PI4K2A 相互作用,而且这种相互作用对于自噬体与溶酶体融合非常重要。在这里,我们确定了 PI4K2A 催化结构域中包含 GABARAP 相互作用基序(GIM)的 7 个氨基酸片段。该片段位于一个暴露的环中,而该环在哺乳动物的另一种 II 型 PI 4-kinase PI4K2B 中并不保守,这就解释了 GABARAP 与 PI4K2A 异构体结合的特异性。PI4K2A GIM 的突变抑制了 GABARAP 的结合以及 PI4K2A 介导的细胞质 GABARAP 到亚细胞器的招募。我们进一步发现,GABARAP 能与单磷酸化磷酸肌酸、PI3P、PI4P 和 PI5P 结合,这就提出了一种可能性,即这些脂质有助于产生结合能,从而推动 GABARAP 与膜上蛋白的相互作用。
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Identification of the GABARAP binding determinant in PI4K2A.

GABARAP is a member of the ATG8 family of ubiquitin-like autophagy related proteins. It was initially discovered as a facilitator of GABA-A receptor translocation to the plasma membrane and has since been shown to promote the intracellular transport of a variety of other proteins under non-autophagic conditions. We and others have shown that GABARAP interacts with the Type II phosphatidylinositol 4-kinase, PI4K2A, and that this interaction is important for autophagosome-lysosome fusion. Here, we identify a 7-amino acid segment within the PI4K2A catalytic domain that contains the GABARAP interaction motif (GIM). This segment resides in an exposed loop that is not conserved in the other mammalian Type II PI 4-kinase, PI4K2B, explaining the specificity of GABARAP binding to the PI4K2A isoform. Mutation of the PI4K2A GIM inhibits GABARAP binding and PI4K2A-mediated recruitment of cytosolic GABARAP to subcellular organelles. We further show that GABARAP binds to mono-phosphorylated phosphoinositides, PI3P, PI4P, and PI5P, raising the possibility that these lipids contribute to the binding energies that drive GABARAP-protein interactions on membranes.

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来源期刊
Bioscience Reports
Bioscience Reports 生物-细胞生物学
CiteScore
8.50
自引率
0.00%
发文量
380
审稿时长
6-12 weeks
期刊介绍: Bioscience Reports provides a home for sound scientific research in all areas of cell biology and molecular life sciences. Since 2012, Bioscience Reports has been fully Open Access and publishes all papers under the liberal CC BY licence, giving the life science community quality research to share and discuss.Content before 2012 is subscription-only, and is accessible via archive purchase. Articles are assessed on soundness, providing a home for valid findings and data. We welcome papers that span disciplines (e.g. chemistry, medicine), including papers describing: -new methodologies -tools and reagents to probe biological questions -mechanistic details -disease mechanisms -metabolic processes and their regulation -structure and function -bioenergetics
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