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引用次数: 0
摘要
撤回：C. Shi, T. Liu, J. Chi, H. Luo, Z. Wu, B. Xiong, S. Liu, Y. Zeng, "LINC00339 promotes gastric cancer progression by elevating DCP1A expression via inhibiting miR-377-3p," Journal of Cellular Physiology 234, no. 12 (2019): 23667-23674, https://doi.org/10.1002/jcp.28934.上述文章于 2019 年 6 月 12 日在线发表于 Wiley Online Library (wileyonlinelibrary.com)，经作者、期刊主编亚历山大-哈奇森（Alexander Hutchison）和 Wiley Periodicals LLC 协议撤回。撤稿是应作者要求达成的，原因是文章中提供的数据令人担忧。期刊在随后进行的调查中发现，文章中的结果与描述的实验方法存在若干不一致之处。具体来说，发现实验方法缺乏或无法获得支持数据，使读者无法理解实验。此外，所提供的原始数据也不能完全支持所展示的结果。因此，编辑认为这篇文章的结论无效。
RETRACTION: LINC00339 promotes gastric cancer progression by elevating DCP1A expression via inhibiting miR-377-3p
RETRACTION: C. Shi, T. Liu, J. Chi, H. Luo, Z. Wu, B. Xiong, S. Liu, Y. Zeng, “LINC00339 promotes gastric cancer progression by elevating DCP1A expression via inhibiting miR-377-3p,” Journal of Cellular Physiology 234, no. 12 (2019): 23667-23674, https://doi.org/10.1002/jcp.28934.
The above article, published online on 12 June 2019 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the authors; the journal Editor-in-Chief, Alexander Hutchison; and Wiley Periodicals LLC.
The retraction has been agreed upon the authors' request due to concerns related to the data presented in the article. In the following investigation performed by the journal, several inconsistencies between results presented and experimental methods described were found. Specifically, the experimental methods were found to lack or have unavailable supporting data, making the experiments not comprehensible to readers. Additionally, the raw data provided do not entirely support the results presented. Accordingly, the conclusions of this article are considered invalid by the editors.
期刊介绍：
The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
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