全基因组DNA甲基化分析揭示了莫亚莫亚病的低甲基化变异性

IF 3.8 2区 医学 Q1 CLINICAL NEUROLOGY Translational Stroke Research Pub Date : 2024-10-02 DOI:10.1007/s12975-024-01299-w
Kikutaro Tokairin, Masaki Ito, Alex G Lee, Mario Teo, Shihao He, Michelle Y Cheng, Gary K Steinberg
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引用次数: 0

摘要

莫亚莫亚病(MMD)是一种慢性脑血管疾病,可导致中风和神经功能障碍。鉴于该病多为散发性,且基因与环境之间的相互作用在各种疾病中起着重要作用,我们对MMD的表观遗传修饰进行了研究。我们使用 Illumina 850 K Methylation EPIC BeadChip 对两个不同种族的成年女性队列进行了全基因组 DNA 甲基化分析:一个非亚洲队列(13 名 MMD 患者和 7 名健康对照)和一个亚洲队列(14 名 MMD 患者和 3 名健康对照)。此外,我们还加入了一个外部男女队列(女性:5 名麻风病患者和 5 名健康对照组;男性:5 名麻风病患者和 5 名健康对照组)进行验证。我们的研究结果表明,在 MMD 女性队列中,MMD 患者与健康对照组之间的 DNA 甲基化变异性非常低。在非亚洲人队列中,只有 6 个探针的变异性增加,而 647 个探针的变异性降低。同样,在亚裔队列中,MMD 组的所有 2845 个探针的甲基化变异性也有所降低。随后的分析表明,这些差异探针位于参与甲基化和转录、DNA 修复、细胞骨架重塑、自然杀伤细胞信号传导、细胞生长和迁移等关键生物过程的基因上。这些发现标志着首次在任何疾病中观察到低甲基化变异性,与在其他疾病中观察到的高变异性形成鲜明对比。MMD患者甲基化变异性的降低可能会阻碍患者对环境变化(如血流动力学压力)的适应性,从而影响血管稳态并导致MMD病理变化。这些发现为MMD的发病机制和潜在治疗策略提供了新的见解。
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Genome-Wide DNA Methylation Profiling Reveals Low Methylation Variability in Moyamoya Disease.

Moyamoya disease (MMD) is a chronic cerebrovascular disorder that can lead to stroke and neurological dysfunctions. Given the largely sporadic nature and the role of gene-environment interactions in various diseases, we examined epigenetic modifications in MMD. We performed genome-wide DNA methylation using Illumina 850 K Methylation EPIC BeadChip, in two racially distinct adult female cohorts: a non-Asian cohort (13 MMD patients and 7 healthy controls) and an Asian cohort (14 MMD patients and 3 healthy controls). An additional external cohort with both sexes (females: 5 MMD patients and 5 healthy controls, males: 5 MMD patients and 5 healthy controls) was included for validation. Our findings revealed strikingly low DNA methylation variability between MMD patients and healthy controls, in both MMD female cohorts. In the non-Asian cohort, only 6 probes showed increased variability versus 647 probes that showed decreased variability. Similarly, in the Asian cohort, the MMD group also displayed a reduced methylation variability across all 2845 probes. Subsequent analysis showed that these differentially variable probes are located on genes involved in key biological processes such as methylation and transcription, DNA repair, cytoskeletal remodeling, natural killer cell signaling, cellular growth, and migration. These findings mark the first observation of low methylation variability in any disease, contrasting with the high variability observed in other disorders. This reduced methylation variability in MMD may hinder patients' adaptability to environmental shifts, such as hemodynamic stress, thereby influencing vascular homeostasis and contributing to MMD pathology. These findings offer new insights into the mechanisms of MMD and potential treatment strategies.

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来源期刊
Translational Stroke Research
Translational Stroke Research CLINICAL NEUROLOGY-NEUROSCIENCES
CiteScore
13.80
自引率
4.30%
发文量
130
审稿时长
6-12 weeks
期刊介绍: Translational Stroke Research covers basic, translational, and clinical studies. The Journal emphasizes novel approaches to help both to understand clinical phenomenon through basic science tools, and to translate basic science discoveries into the development of new strategies for the prevention, assessment, treatment, and enhancement of central nervous system repair after stroke and other forms of neurotrauma. Translational Stroke Research focuses on translational research and is relevant to both basic scientists and physicians, including but not restricted to neuroscientists, vascular biologists, neurologists, neuroimagers, and neurosurgeons.
期刊最新文献
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