内皮细胞是长 COVID 的旁分泌介质

IF 9.4 Q1 CARDIAC & CARDIOVASCULAR SYSTEMS Nature cardiovascular research Pub Date : 2024-10-14 DOI:10.1038/s44161-024-00551-8
Simon R. Foster, James E. Hudson
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引用次数: 0

摘要

长期 COVID 导致多器官功能障碍的分子机制尚不清楚,尤其是心血管系统。研究发现,内皮反应和信号传导在驱动功能障碍方面起着关键作用。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

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Endothelial cells as paracrine mediators of long COVID
The molecular mechanisms that underpin the multi-organ dysfunction in long COVID are unknown, particularly within the cardiovascular system. Research finds a critical role for endothelial responses and signaling in driving dysfunction.
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