返回:MATN1-AS1通过作为miR-200b/c/429的ceRNA来调节CHD1的表达,从而促进胶质瘤的进展。

IF 5.9 1区 生物学 Q2 CELL BIOLOGY Cell Proliferation Pub Date : 2024-10-20 DOI:10.1111/cpr.13767
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引用次数: 0

摘要

J.Zhu , WT.Gu , and C. Yu , "MATN1-AS1 Promotes Glioma Progression by Functioning as ceRNA of miR-200b/c/429 to Regulate CHD1 Expression," Cell Proliferation 53, no. 1 (2020): e12700. https://doi.org/10.1111/cpr.12700.上述文章于2019年10月30日在线发表于《威利在线图书馆》(wileyonlinelibrary.com),经作者、期刊副主编林云峰和John Wiley & Sons Ltd.三方协商,已被撤稿。作者联系了期刊并报告说,他们发现数字中的错误影响了文章结果的有效性,因此要求撤稿。此外,第三方报告发现图 2E 和图 2C 与不同作者的其他文章重复,每篇文章描述的实验条件也不同。在进一步通信后,作者承认了这些重复,并表示他们无法确认数据的真实性。由于不同文章中的重复图片从根本上损害了文章中的结论和结果,因此作者同意撤回文章。
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RETRACTION: MATN1-AS1 promotes glioma progression by functioning as ceRNA of miR-200b/c/429 to regulate CHD1 expression.

J. Zhu , WT. Gu , and C. Yu , "MATN1-AS1 Promotes Glioma Progression by Functioning as ceRNA of miR-200b/c/429 to Regulate CHD1 Expression," Cell Proliferation 53, no. 1 (2020): e12700. https://doi.org/10.1111/cpr.12700. The above article, published online on 30 October 2019, in Wiley Online Library (wileyonlinelibrary.com), and has been retracted by agreement between the authors; the journal Deputy Editor, Yunfeng Lin; and John Wiley & Sons Ltd. The authors contacted the journal and reported that they had detected mistakes in the figures that compromised the validity of the article's results and requested a retraction. In addition, a report from a third party found duplications between Figures 2E and 2C with other articles by different authors, each of which describe different experimental conditions. Following further correspondence, the authors acknowledged these duplications and have stated that they cannot confirm the authenticity of the data. The retraction has been agreed to because the duplications of images across different articles fundamentally compromises the conclusions and results presented in the article.

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来源期刊
Cell Proliferation
Cell Proliferation 生物-细胞生物学
CiteScore
14.80
自引率
2.40%
发文量
198
审稿时长
1 months
期刊介绍: Cell Proliferation Focus: Devoted to studies into all aspects of cell proliferation and differentiation. Covers normal and abnormal states. Explores control systems and mechanisms at various levels: inter- and intracellular, molecular, and genetic. Investigates modification by and interactions with chemical and physical agents. Includes mathematical modeling and the development of new techniques. Publication Content: Original research papers Invited review articles Book reviews Letters commenting on previously published papers and/or topics of general interest By organizing the information in this manner, readers can quickly grasp the scope, focus, and publication content of Cell Proliferation.
期刊最新文献
Direct reprogramming of fibroblasts into spiral ganglion neurons by defined transcription factors. The apoptotic and anti-proliferative effects of Neosetophomone B in T-cell acute lymphoblastic leukaemia via PI3K/AKT/mTOR pathway inhibition. Synergy between pluripotent stem cell-derived macrophages and self-renewing macrophages: Envisioning a promising avenue for the modelling and cell therapy of infectious diseases. Predicting tumour resistance to paclitaxel and carboplatin utilising genome-wide screening in haploid human embryonic stem cells. Issue Information
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