返回:丹参酮 I 通过使 PI3K/AKT/mTOR 通路失活,诱导细胞凋亡和自噬,从而减轻卵巢癌的恶性生物学特性。

IF 5.9 1区 生物学 Q2 CELL BIOLOGY Cell Proliferation Pub Date : 2024-10-23 DOI:10.1111/cpr.13768
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引用次数: 0

摘要

J.Zhou , Y.-y. Jiang , H. Chen , Y.-c.Jiang , H. Chen , Y.-c. Wu , and L. Zhang , "Tanshinone I Attenuates Ovarian Cancer by Inducing Apoptosis and Aut Aut.-y.Wu , and L. Zhang , "Tanshinone I Attenuates the Malignant Biological Properties of Ovarian Cancer by Inducing Apoptosis and Autophagy via the Inactivation of PI3K/AKT/mTOR Pathway," Cell Proliferation 53, no. 2 (2020): e12739. https://doi.org/10.1111/cpr.12739.上述文章于 2019 年 12 月 09 日在线发表于 Wiley Online Library(wileyonlinelibrary.com),经期刊副主编林云峰与 John Wiley & Sons Ltd.(约翰-威利父子有限公司)协商,已被撤稿。第三方联系了出版商,报告称在不同作者组的多篇不同文章中发现了重复图像,每篇文章都描述了不同的实验条件。重复图像如下:图 1C 中的图像重复出现在 Wang 等人 2019 年的文章(https://doi.org/10.2147/OTT.S221161)中,该文章是在《细胞增殖》发表本文之前提交和发表的。此外,图 1C、1D、2C、3B、3D、4C、4D 和 5D 中的图像在本文之后发表的不同作者小组的许多其他文章中被发现重复。作者就这些问题回复了出版商的询问,并分享了标注为图 1-6 的原始图片,但没有分享图 1 中使用的细胞增殖实验的原始图片。编辑审查了这一证据,发现它不能正确解释不同文章中的重复。由于不同文章中的图片重复从根本上影响了文章的结论和结果,因此同意撤稿。作者不同意撤稿。
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RETRACTION: Tanshinone I attenuates the malignant biological properties of ovarian cancer by inducing apoptosis and autophagy via the inactivation of PI3K/AKT/mTOR pathway.

J. Zhou , Y.-y. Jiang , H. Chen , Y.-c. Wu , and L. Zhang , "Tanshinone I Attenuates the Malignant Biological Properties of Ovarian Cancer by Inducing Apoptosis and Autophagy via the Inactivation of PI3K/AKT/mTOR Pathway," Cell Proliferation 53, no. 2 (2020): e12739. https://doi.org/10.1111/cpr.12739. The above article, published online on 09 December 2019, in Wiley Online Library (wileyonlinelibrary.com), and has been retracted by agreement between the journal Deputy Editor, Yunfeng Lin; and John Wiley & Sons Ltd. A third party contacted the publisher to report that duplicated images had been detected in multiple different articles by different author groups, each of which described different experimental conditions. The image duplications are listed as follows: Images in Figure 1C were duplicated in Wang et al. 2019 (https://doi.org/10.2147/OTT.S221161), which was submitted and published prior to the publication of this article in Cell Proliferation. In addition, duplicate images from Figures 1C, 1D, 2C, 3B, 3D, 4C, 4D, and 5D were detected in many other articles by different author groups that had been published subsequent to this article. The authors responded to an inquiry by the publisher regarding these concerns and shared what were labeled as original images for Figures 1-6 but did not share the original images for the cell proliferation assays that had been used in Figure 1. The editors reviewed this evidence and found that it did not properly explain the duplications across different articles. The retraction has been agreed to because the duplications of images across different articles fundamentally compromises the conclusions and results presented in the article. The authors disagree with the retraction.

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来源期刊
Cell Proliferation
Cell Proliferation 生物-细胞生物学
CiteScore
14.80
自引率
2.40%
发文量
198
审稿时长
1 months
期刊介绍: Cell Proliferation Focus: Devoted to studies into all aspects of cell proliferation and differentiation. Covers normal and abnormal states. Explores control systems and mechanisms at various levels: inter- and intracellular, molecular, and genetic. Investigates modification by and interactions with chemical and physical agents. Includes mathematical modeling and the development of new techniques. Publication Content: Original research papers Invited review articles Book reviews Letters commenting on previously published papers and/or topics of general interest By organizing the information in this manner, readers can quickly grasp the scope, focus, and publication content of Cell Proliferation.
期刊最新文献
Targeting Hsp90α to inhibit HMGB1-mediated renal inflammation and fibrosis. Direct reprogramming of fibroblasts into spiral ganglion neurons by defined transcription factors. The apoptotic and anti-proliferative effects of Neosetophomone B in T-cell acute lymphoblastic leukaemia via PI3K/AKT/mTOR pathway inhibition. Synergy between pluripotent stem cell-derived macrophages and self-renewing macrophages: Envisioning a promising avenue for the modelling and cell therapy of infectious diseases. Predicting tumour resistance to paclitaxel and carboplatin utilising genome-wide screening in haploid human embryonic stem cells.
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