利用数学建模描述癌细胞对周期性缺氧的反应

IF 2 4区 数学 Q2 BIOLOGY Bulletin of Mathematical Biology Pub Date : 2024-11-06 DOI:10.1007/s11538-024-01359-0
Giulia L Celora, Ruby Nixson, Joe M Pitt-Francis, Philip K Maini, Helen M Byrne
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引用次数: 0

摘要

体内观察结果表明,肿瘤内的氧含量可在快慢时间尺度上波动。因此,癌细胞会周期性地暴露在病理低氧水平下;这种现象被称为周期性缺氧。然而,人们对癌细胞对周期性缺氧而非持续性缺氧的反应和适应知之甚少。此外,现有的体外周期性缺氧模型无法捕捉到体内生长的肿瘤复杂而多变的氧动态。数学模型有助于克服当前实验的局限性,并通过预测细胞对各种循环动态的反应,为肿瘤循环缺氧生物学提供新的见解。我们开发了一个基于个体的模型,以研究暴露于周期性缺氧后,癌细胞的细胞周期进展和细胞命运决定是如何改变的。我们的模型可以模拟标准的体外实验,如克隆生成实验和细胞周期实验,从而有效筛选细胞在各种循环缺氧条件下的反应。模拟结果表明,根据氧波动的动态变化,同一细胞系对周期性缺氧的反应会明显不同。我们还利用模型研究了细胞周期检查点激活和损伤修复的变化对细胞周期性缺氧反应的影响。我们的模拟结果表明,周期性缺氧可促进血管肿瘤内细胞损伤修复活动的异质性。
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Characterising Cancer Cell Responses to Cyclic Hypoxia Using Mathematical Modelling.

In vivo observations show that oxygen levels in tumours can fluctuate on fast and slow timescales. As a result, cancer cells can be periodically exposed to pathologically low oxygen levels; a phenomenon known as cyclic hypoxia. Yet, little is known about the response and adaptation of cancer cells to cyclic, rather than, constant hypoxia. Further, existing in vitro models of cyclic hypoxia fail to capture the complex and heterogeneous oxygen dynamics of tumours growing in vivo. Mathematical models can help to overcome current experimental limitations and, in so doing, offer new insights into the biology of tumour cyclic hypoxia by predicting cell responses to a wide range of cyclic dynamics. We develop an individual-based model to investigate how cell cycle progression and cell fate determination of cancer cells are altered following exposure to cyclic hypoxia. Our model can simulate standard in vitro experiments, such as clonogenic assays and cell cycle experiments, allowing for efficient screening of cell responses under a wide range of cyclic hypoxia conditions. Simulation results show that the same cell line can exhibit markedly different responses to cyclic hypoxia depending on the dynamics of the oxygen fluctuations. We also use our model to investigate the impact of changes to cell cycle checkpoint activation and damage repair on cell responses to cyclic hypoxia. Our simulations suggest that cyclic hypoxia can promote heterogeneity in cellular damage repair activity within vascular tumours.

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来源期刊
CiteScore
3.90
自引率
8.60%
发文量
123
审稿时长
7.5 months
期刊介绍: The Bulletin of Mathematical Biology, the official journal of the Society for Mathematical Biology, disseminates original research findings and other information relevant to the interface of biology and the mathematical sciences. Contributions should have relevance to both fields. In order to accommodate the broad scope of new developments, the journal accepts a variety of contributions, including: Original research articles focused on new biological insights gained with the help of tools from the mathematical sciences or new mathematical tools and methods with demonstrated applicability to biological investigations Research in mathematical biology education Reviews Commentaries Perspectives, and contributions that discuss issues important to the profession All contributions are peer-reviewed.
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