更正:Met受体酪氨酸激酶信号的抑制通过激活p38 MAPK通路增强胶质瘤细胞系对CDDP的化学敏感性。

IF 5.3 2区 医学 Q1 ONCOLOGY Molecular Cancer Therapeutics Pub Date : 2025-01-02 DOI:10.1158/1535-7163.MCT-24-0974
Xiuqin Lou, Qibing Zhou, Ying Yin, Cheng Zhou, Yan Shen
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Correction: Inhibition of the Met Receptor Tyrosine Kinase Signaling Enhances the Chemosensitivity of Glioma Cell Lines to CDDP Through Activation of p38 MAPK Pathway.
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来源期刊
CiteScore
11.20
自引率
1.80%
发文量
331
审稿时长
3 months
期刊介绍: Molecular Cancer Therapeutics will focus on basic research that has implications for cancer therapeutics in the following areas: Experimental Cancer Therapeutics, Identification of Molecular Targets, Targets for Chemoprevention, New Models, Cancer Chemistry and Drug Discovery, Molecular and Cellular Pharmacology, Molecular Classification of Tumors, and Bioinformatics and Computational Molecular Biology. The journal provides a publication forum for these emerging disciplines that is focused specifically on cancer research. Papers are stringently reviewed and only those that report results of novel, timely, and significant research and meet high standards of scientific merit will be accepted for publication.
期刊最新文献
Identification of a TNIK-CDK9 axis as a targetable strategy for platinum-resistant ovarian cancer. LIG1 is a synthetic lethal target in BRCA1 mutant cancers. VAX014 Activates Tumor-Intrinsic STING and RIG-I to Promote the Development of Antitumor Immunity. Co-blocking TIGIT and PVRIG using a novel bispecific antibody enhances anti-tumor immunity. PI3K/mTOR dual inhibitor GSK458 and arsenic trioxide exert synergistic anti-tumor effects against ovarian clear cell carcinoma.
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