线粒体、溶酶体和其他细胞器之间的相互作用如何预防或促进干性老年性黄斑变性。

IF 3 2区 医学 Q1 OPHTHALMOLOGY Experimental eye research Pub Date : 2024-12-22 DOI:10.1016/j.exer.2024.110219
Aparna Lakkaraju, Patricia Boya, Marie Csete, Deborah A Ferrington, James B Hurley, Alfredo A Sadun, Peng Shang, Ruchi Sharma, Debasish Sinha, Marius Ueffing, Susan E Brockerhoff
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引用次数: 0

摘要

细胞器如线粒体、溶酶体、过氧化物酶体和内质网形成高度动态的细胞网络,并通过物理接触部位交换信息。虽然每个细胞器都有独特的功能,但这种细胞器间的相互作用有助于维持细胞的稳态。年龄相关性黄斑变性(AMD)是一种毁灭性致盲疾病,与线粒体功能障碍、氧化应激和视网膜色素上皮(RPE)细胞碎片清除减少密切相关。然而,这些是如何发生的,以及它们如何与RPE和潜在的光感受器的细胞器功能相关,是AMD生物学中根本的,未解决的问题。在这里,我们报告了2024年瑞安黄斑研究倡议(RIMR)的“线粒体,溶酶体和其他细胞器相互作用”任务组的讨论。我们的研究小组专注于了解细胞器在维持RPE和光感受器内稳态方面的相互作用,这是如何促进AMD的,并确定这些途径在哪里可能成为治疗的靶点。
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How crosstalk between mitochondria, lysosomes, and other organelles can prevent or promote dry age-related macular degeneration.

Organelles such as mitochondria, lysosomes, peroxisomes, and the endoplasmic reticulum form highly dynamic cellular networks and exchange information through sites of physical contact. While each organelle performs unique functions, this inter-organelle crosstalk helps maintain cell homeostasis. Age-related macular degeneration (AMD) is a devastating blinding disease strongly associated with mitochondrial dysfunction, oxidative stress, and decreased clearance of cellular debris in the retinal pigment epithelium (RPE). However, how these occur, and how they relate to organelle function both with the RPE and potentially the photoreceptors are fundamental, unresolved questions in AMD biology. Here, we report the discussions of the "Mitochondria, Lysosomes, and other Organelle Interactions" task group of the 2024 Ryan Initiative for Macular Research (RIMR). Our group focused on understanding the interplay between cellular organelles in maintaining homeostasis in the RPE and photoreceptors, how this could be derailed to promote AMD, and identifying where these pathways could potentially be targeted therapeutically.

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来源期刊
Experimental eye research
Experimental eye research 医学-眼科学
CiteScore
6.80
自引率
5.90%
发文量
323
审稿时长
66 days
期刊介绍: The primary goal of Experimental Eye Research is to publish original research papers on all aspects of experimental biology of the eye and ocular tissues that seek to define the mechanisms of normal function and/or disease. Studies of ocular tissues that encompass the disciplines of cell biology, developmental biology, genetics, molecular biology, physiology, biochemistry, biophysics, immunology or microbiology are most welcomed. Manuscripts that are purely clinical or in a surgical area of ophthalmology are not appropriate for submission to Experimental Eye Research and if received will be returned without review.
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