CDK5与MST2相互作用并调节Hippo信号通路。

IF 2.8 4区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY FEBS Open Bio Pub Date : 2024-12-30 DOI:10.1002/2211-5463.13962
Mehak Passi, Jan B Stöckl, Thomas Fröhlich, Simone Moser, Angelika M Vollmar, Stefan Zahler
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引用次数: 0

摘要

MST2 (STK3)是Hippo信号通路的一个主要上游激酶,Hippo信号通路是一个进化保守的调节器官大小、自我更新和组织稳态的途径。其下游效应因子是转录调控因子YAP和TAZ。这一途径受到多种因素的调控,如基质刚度或细胞-细胞接触。利用酵母双杂交筛选,我们发现了MST2和CDK5激酶之间的一种新的相互作用,我们通过共免疫沉淀实验进一步证实了这一点。周期蛋白依赖性激酶5 (CDK5)是周期蛋白依赖性激酶家族中不寻常的成员,参与肿瘤生长和血管生成。尽管CDK5和Hippo之间的联系先前已经被假设,但其作用方式仍然难以捉摸。在这里,我们发现敲低CDK5导致YAP转录活性降低,并且CDK5影响Hippo上游激酶LATS1的磷酸化水平。此外,磷酸化蛋白质组学方法显示,CDK5干扰另一个上游激酶DLG5的磷酸化,DLG5调节Hippo通路。因此,CDK5似乎作为整合Hippo通路和其他信号级联的信号中枢。这些相互作用可能对CDK5抑制剂的使用具有重要意义,CDK5抑制剂已经在临床用于肿瘤疾病。
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CDK5 interacts with MST2 and modulates the Hippo signalling pathway.

MST2 (STK3) is a major upstream kinase in the Hippo signalling pathway, an evolutionary conserved pathway in regulation of organ size, self-renewal and tissue homeostasis. Its downstream effectors are the transcriptional regulators YAP and TAZ. This pathway is regulated by a variety of factors, such as substrate stiffness or cell-cell contacts. Using a yeast two-hybrid screen, we detected a novel interaction between the kinases MST2 and CDK5, which we further confirmed by co-immunoprecipitation experiments. Cyclin-dependent kinase 5 (CDK5) is an unusual member of the family of cyclin-dependent kinases, involved in tumour growth and angiogenesis. Although a link between CDK5 and Hippo has been previously postulated, the mode of action is still elusive. Here, we show that knockdown of CDK5 causes reduced transcriptional activity of YAP and that CDK5 influences the phosphorylation levels of the Hippo upstream kinase LATS1. Moreover, a phosphoproteomics approach revealed that CDK5 interferes with the phosphorylation of DLG5, another upstream kinase, which regulates the Hippo pathway. Hence, CDK5 seems to act as a signalling hub for integrating the Hippo pathway and other signalling cascades. These interactions might have important implications for the use of CDK5 inhibitors, which are already in clinical use for tumour diseases.

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来源期刊
FEBS Open Bio
FEBS Open Bio BIOCHEMISTRY & MOLECULAR BIOLOGY-
CiteScore
5.10
自引率
0.00%
发文量
173
审稿时长
10 weeks
期刊介绍: FEBS Open Bio is an online-only open access journal for the rapid publication of research articles in molecular and cellular life sciences in both health and disease. The journal''s peer review process focuses on the technical soundness of papers, leaving the assessment of their impact and importance to the scientific community. FEBS Open Bio is owned by the Federation of European Biochemical Societies (FEBS), a not-for-profit organization, and is published on behalf of FEBS by FEBS Press and Wiley. Any income from the journal will be used to support scientists through fellowships, courses, travel grants, prizes and other FEBS initiatives.
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