CD4+ T细胞重新连接肉芽肿细胞和调节网络,促进Mtb再感染后的免疫调节

IF 25.5 1区 医学 Q1 IMMUNOLOGY Immunity Pub Date : 2025-01-15 DOI:10.1016/j.immuni.2025.01.001
Joshua D. Bromley, Sharie Keanne C. Ganchua, Sarah K. Nyquist, Pauline Maiello, Michael Chao, H. Jacob Borish, Mark Rodgers, Jaime Tomko, Kara Kracinovsky, Douaa Mugahid, Son Nguyen, Qianchang Dennis Wang, Jacob M. Rosenberg, Edwin C. Klein, Hannah P. Gideon, Roisin Floyd-O’Sullivan, Bonnie Berger, Charles A. Scanga, Philana Ling Lin, Sarah M. Fortune, JoAnne L. Flynn
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引用次数: 0

摘要

(免疫57,2380-2398.e1-e6;2024年10月8日)
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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CD4+ T cells re-wire granuloma cellularity and regulatory networks to promote immunomodulation following Mtb reinfection
(Immunity 57, 2380–2398.e1–e6; October 8, 2024)
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来源期刊
Immunity
Immunity 医学-免疫学
CiteScore
49.40
自引率
2.20%
发文量
205
审稿时长
6 months
期刊介绍: Immunity is a publication that focuses on publishing significant advancements in research related to immunology. We encourage the submission of studies that offer groundbreaking immunological discoveries, whether at the molecular, cellular, or whole organism level. Topics of interest encompass a wide range, such as cancer, infectious diseases, neuroimmunology, autoimmune diseases, allergies, mucosal immunity, metabolic diseases, and homeostasis.
期刊最新文献
The endogenous antigen-specific CD8+ T cell repertoire is composed of unbiased and biased clonotypes with differential fate commitments A bladder-blood immune barrier constituted by suburothelial perivascular macrophages restrains uropathogen dissemination OAS cross-activates RNase L intercellularly through cell-to-cell transfer of 2-5A to spread innate immunity The kinase RIPK3 promotes neuronal survival by suppressing excitatory neurotransmission during central nervous system viral infection Cutting to the chase: Pruning alloreactive T cells
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