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Doping cDC1s for tolerance 掺杂cDC1s以增强耐受性
IF 32.4 1区 医学 Q1 IMMUNOLOGY Pub Date : 2026-02-10 DOI: 10.1016/j.immuni.2026.01.020
Victor Bosteels, Sophie Janssens
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引用次数: 0
TCRAFT: A Rosetta Stone for T cell receptors T细胞受体的罗塞塔石碑
IF 32.4 1区 医学 Q1 IMMUNOLOGY Pub Date : 2026-02-10 DOI: 10.1016/j.immuni.2026.01.024
Christopher A. Polera, Alex M. Jaeger
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引用次数: 0
Chromatin-mediated anticipatory control of type I interferon production in plasmacytoid dendritic cells 浆细胞样树突状细胞中染色质介导的I型干扰素产生的预期控制
IF 32.4 1区 医学 Q1 IMMUNOLOGY Pub Date : 2026-02-10 DOI: 10.1016/j.immuni.2025.12.005
Nicholas M. Adams, Aleksandra Galitsyna, Ioanna Tiniakou, Eduardo Esteva, Ai C. Ra, Simon Ullrich, Stephen T. Yeung, Yanjun Tan, Joseph N. Pucella, Igor Dolgalev, David E. Levy, Kamal M. Khanna, Irina Solovei, Leonid A. Mirny, Boris Reizis
{"title":"Chromatin-mediated anticipatory control of type I interferon production in plasmacytoid dendritic cells","authors":"Nicholas M. Adams, Aleksandra Galitsyna, Ioanna Tiniakou, Eduardo Esteva, Ai C. Ra, Simon Ullrich, Stephen T. Yeung, Yanjun Tan, Joseph N. Pucella, Igor Dolgalev, David E. Levy, Kamal M. Khanna, Irina Solovei, Leonid A. Mirny, Boris Reizis","doi":"10.1016/j.immuni.2025.12.005","DOIUrl":"https://doi.org/10.1016/j.immuni.2025.12.005","url":null,"abstract":"","PeriodicalId":13269,"journal":{"name":"Immunity","volume":"18 1","pages":""},"PeriodicalIF":32.4,"publicationDate":"2026-02-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"146152843","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Hormonal rewiring of immunity during dietary restriction ensures host defense and systemic glucose conservation 在饮食限制期间,免疫系统的激素重组确保了宿主的防御和全身葡萄糖的保存
IF 32.4 1区 医学 Q1 IMMUNOLOGY Pub Date : 2026-02-10 DOI: 10.1016/j.immuni.2026.01.003
Luisa Menezes-Silva, Mingeum Jeong, Charles Carr, Riley M. Schneider, Silvia Pires, Ana C. Codo, Jazib Uddin, Alexander Grier, Randy S. Longman, Niels Olsen Saraiva Camara, David Artis, Seong-Ji Han, Nicholas Collins
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引用次数: 0
Shedding light on interventions for brain aging 揭示了大脑衰老的干预措施
IF 32.4 1区 医学 Q1 IMMUNOLOGY Pub Date : 2026-02-10 DOI: 10.1016/j.immuni.2026.01.021
Juan Zhang, Qiang Liu
Immunotherapeutic approaches to brain aging remain largely preclinical and in early translational stages, and they have focused mostly on modulating innate immunity. In this issue of Immunity, Negredo et al. identify T cells bearing exhaustion-like signatures as a hallmark of brain aging and reveal the beneficial effects of an engineered IL-10 variant that functionally uncouples pro- and anti-inflammatory signaling in microglia.
脑衰老的免疫治疗方法仍主要停留在临床前和早期转化阶段,它们主要集中在调节先天免疫。在这一期的《免疫》杂志上,Negredo等人发现T细胞携带衰竭样信号是大脑衰老的标志,并揭示了一种工程IL-10变体的有益作用,该变体在功能上解耦了小胶质细胞中的促炎和抗炎信号。
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引用次数: 0
Microglia makeover: On-demand control panel revamp 小胶质细胞改造:按需控制面板改造
IF 32.4 1区 医学 Q1 IMMUNOLOGY Pub Date : 2026-02-10 DOI: 10.1016/j.immuni.2026.01.007
Takahiro Masuda
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引用次数: 0
Cystine fuels ILC2s to survive lung inflammation 胱氨酸促进ILC2s在肺部炎症中存活
IF 32.4 1区 医学 Q1 IMMUNOLOGY Pub Date : 2026-02-10 DOI: 10.1016/j.immuni.2026.01.018
Yuchao Jing, Jie Zhou
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引用次数: 0
Influenza hijacks myeloid cells to inflict type-I interferon-fueled damage in the heart 流感劫持骨髓细胞,在心脏造成i型干扰素引发的损伤
IF 32.4 1区 医学 Q1 IMMUNOLOGY Pub Date : 2026-02-09 DOI: 10.1016/j.immuni.2025.12.011
Jeffrey Downey, Ana Oliveira-Coelho, Máté G. Kiss, Gabriel Laghlali, Alexander Leunig, Emir Radkevich, Laszlo Halasz, Martin Umali, Joana Ferreira da Silva, Madeline L. Eller, Matteo Gianeselli, Magdalena M. Żak, Haley E. Randolph, Gabriel Caumartin, Wolfram C. Poller, Henrike Janssen, Laura L. Koekkoek, Jamshid Abdul-Ghafar, Pacific Huynh, Darwin D’Souza, Vladimir Roudko, Sheqouia Nauta, Jazz Munitz, Xisheng Li, Thomas Rathner, Ziche Chen, Anh Phan, Abigail Glick, Katarzyna Cialowicz, Zhihong Chen, Seunghee Kim-Schulze, Seonghun Yoon, Matthias Nahrendorf, Susmita Sahoo, Miriam Merad, Adolfo García-Sastre, Cameron S. McAlpine, Gustav J. Strijkers, Viviana Simon, Rachel Brody, Saurabh Mehandru, Zahi A. Fayad, Benjamin P. Kleinstiver, Lior Zangi, Mandy M.T. van Leent, Michael Schotsaert, Filip K. Swirski
Abundant evidence has correlated influenza infection with cardiovascular disease, yet mechanisms linking infection with the heart remain poorly understood. Here, we show that influenza infection damaged the human and murine heart. In mice, we showed that shortly after pulmonary infection, the virus infected a circulating myeloid pro-dendritic cell 3 (pro-DC3) that expressed high concentrations of the chemokine receptor CCR2. The heart, which produces abundant CCL2, preferentially attracted infected pro-DC3. In the myocardium, the virus escaped pro-DC3, infected cardiomyocytes, and triggered production of type-I interferon (IFN-I). Engagement of the IFN-I receptor (IFNAR1) on cardiomyocytes caused tissue damage and compromised heart function. Genetically and therapeutically dampening IFNAR1 exclusively in cardiomyocytes protected the heart while preserving anti-viral immunity in the lung. Our results identify a series of host-pathogen interactions that propagate tissue damage and uncover an axis for intervention to mitigate cardiovascular risk following viral infection.
大量证据表明流感感染与心血管疾病相关,但将感染与心脏联系起来的机制仍然知之甚少。在这里,我们证明流感感染损害了人类和小鼠的心脏。在小鼠中,我们发现在肺部感染后不久,病毒感染了表达高浓度趋化因子受体CCR2的循环髓系前树突状细胞3(前dc3)。产生大量CCL2的心脏优先吸引被感染的原dc3。在心肌中,病毒逃离原dc3,感染心肌细胞,并触发i型干扰素(IFN-I)的产生。IFN-I受体(IFNAR1)对心肌细胞的作用导致组织损伤和心功能受损。基因和治疗上抑制IFNAR1仅在心肌细胞中保护心脏,同时保持肺的抗病毒免疫。我们的研究结果确定了一系列宿主-病原体相互作用,这些相互作用会传播组织损伤,并揭示了一种干预轴,以减轻病毒感染后的心血管风险。
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引用次数: 0
Repression of RIPK1 kinase by INPP5D inhibits expression of diverse proinflammatory mediators and late-onset Alzheimer’s disease risk factors INPP5D抑制RIPK1激酶可抑制多种促炎介质和晚发性阿尔茨海默病危险因素的表达
IF 32.4 1区 医学 Q1 IMMUNOLOGY Pub Date : 2026-02-02 DOI: 10.1016/j.immuni.2026.01.014
Xingxing Xie, Jianping Liu, Wei Liang, Yici Zhang, Xueqi Gong, Shenghao Yuan, Chunting Qi, Maoqing Huang, Linyu Shi, Meiling Hou, Mengmeng Zhang, Wei Liu, Weimin Sun, Yaqi Wu, Cui Li, Ze Cao, Hongyang Jing, Lihui Qian, Jingli Liu, Shufen Yuan, Qiong Wang, Yong Shen, Zhijun Liu, Yunxia Li, Heling Pan, Bing Zhu, Bing Shan, Kaiwen He, Wenyuan Wang, Chengyu Zou, Ying Li, James J. Chou, Junying Yuan
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引用次数: 0
Meningeal B cells: Emerging players at the brain border 脑膜B细胞:脑边界的新兴参与者
IF 32.4 1区 医学 Q1 IMMUNOLOGY Pub Date : 2026-01-30 DOI: 10.1016/j.immuni.2026.01.013
Dianyu Chen, Heping Xu
The meninges, long viewed as passive protective membranes, are now recognized as active immunological interfaces harboring diverse immune populations. Among them, B cells have emerged as dynamic participants in central nervous system (CNS) homeostasis and disorders. Recent studies have identified distinct B cell subsets in the meninges at different developmental and activation stages, including precursors supported by skull marrow-derived progenitors and immunoglobulin A (IgA)⁺ plasma cells influenced by gut microbiota. These meningeal B cells contribute to immune tolerance, barrier protection, and potentially neural development and repair, while also exhibiting context-dependent pathogenic roles in CNS autoimmunity, aging, and neurodegeneration. In this review, we discuss the current knowledge of meningeal B cells and highlight key future research directions aimed at understanding their cellular dynamics, activation and differentiation processes, and the balance between protective and deleterious functions.
脑膜,长期以来被认为是被动的保护膜,现在被认为是活跃的免疫界面窝藏不同的免疫群体。其中,B细胞已成为中枢神经系统(CNS)稳态和失调的动态参与者。最近的研究已经确定了脑膜中处于不同发育和激活阶段的不同B细胞亚群,包括由颅骨骨髓源性祖细胞支持的前体和受肠道微生物群影响的免疫球蛋白A (IgA) +浆细胞。这些脑膜B细胞有助于免疫耐受、屏障保护和潜在的神经发育和修复,同时在中枢神经系统自身免疫、衰老和神经退行性变中也表现出环境依赖性的致病作用。在这篇综述中,我们讨论了目前对脑膜B细胞的认识,并强调了未来的重点研究方向,旨在了解其细胞动力学,激活和分化过程,以及保护和有害功能之间的平衡。
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引用次数: 0
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Immunity
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