{"title":"对“ARID1A下调通过VIM激活和CDH1抑制促进结肠癌细胞增殖和迁移”的修正","authors":"","doi":"10.1111/jcmm.70191","DOIUrl":null,"url":null,"abstract":"<p>Baldi S, Zhang Q, Zhang Z, et al. ARID1A downregulation promotes cell proliferation and migration of colon cancer via VIM activation and CDH1 suppression. <i>J Cell Mol Med.</i> 2022;26:5984-5997. doi:10.1111/jcmm.17590.</p><p>In Salem Baldi et al., the VIM image of HCT116 cells in Figure 5B overlapped the VIM in Figure 5A due to technical error during image preparation. The correct figure is shown below. The authors confirm all results and conclusions of this article remain unchanged.</p>","PeriodicalId":101321,"journal":{"name":"JOURNAL OF CELLULAR AND MOLECULAR MEDICINE","volume":"29 3","pages":""},"PeriodicalIF":4.2000,"publicationDate":"2025-02-11","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/jcmm.70191","citationCount":"0","resultStr":"{\"title\":\"Correction to ‘ARID1A downregulation promotes cell proliferation and migration of colon cancer via VIM activation and CDH1 suppression’\",\"authors\":\"\",\"doi\":\"10.1111/jcmm.70191\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>Baldi S, Zhang Q, Zhang Z, et al. ARID1A downregulation promotes cell proliferation and migration of colon cancer via VIM activation and CDH1 suppression. <i>J Cell Mol Med.</i> 2022;26:5984-5997. doi:10.1111/jcmm.17590.</p><p>In Salem Baldi et al., the VIM image of HCT116 cells in Figure 5B overlapped the VIM in Figure 5A due to technical error during image preparation. The correct figure is shown below. The authors confirm all results and conclusions of this article remain unchanged.</p>\",\"PeriodicalId\":101321,\"journal\":{\"name\":\"JOURNAL OF CELLULAR AND MOLECULAR MEDICINE\",\"volume\":\"29 3\",\"pages\":\"\"},\"PeriodicalIF\":4.2000,\"publicationDate\":\"2025-02-11\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1111/jcmm.70191\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"JOURNAL OF CELLULAR AND MOLECULAR MEDICINE\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1111/jcmm.70191\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"JOURNAL OF CELLULAR AND MOLECULAR MEDICINE","FirstCategoryId":"1085","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1111/jcmm.70191","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Correction to ‘ARID1A downregulation promotes cell proliferation and migration of colon cancer via VIM activation and CDH1 suppression’
Baldi S, Zhang Q, Zhang Z, et al. ARID1A downregulation promotes cell proliferation and migration of colon cancer via VIM activation and CDH1 suppression. J Cell Mol Med. 2022;26:5984-5997. doi:10.1111/jcmm.17590.
In Salem Baldi et al., the VIM image of HCT116 cells in Figure 5B overlapped the VIM in Figure 5A due to technical error during image preparation. The correct figure is shown below. The authors confirm all results and conclusions of this article remain unchanged.
期刊介绍:
The Journal of Cellular and Molecular Medicine serves as a bridge between physiology and cellular medicine, as well as molecular biology and molecular therapeutics. With a 20-year history, the journal adopts an interdisciplinary approach to showcase innovative discoveries.
It publishes research aimed at advancing the collective understanding of the cellular and molecular mechanisms underlying diseases. The journal emphasizes translational studies that translate this knowledge into therapeutic strategies. Being fully open access, the journal is accessible to all readers.