甲基磺酰甲烷通过调节氧化应激介导的骨吸收对老年性骨质流失进展的保护作用。

IF 6.6 2区 医学 Q1 BIOCHEMISTRY & MOLECULAR BIOLOGY Antioxidants Pub Date : 2025-02-13 DOI:10.3390/antiox14020216
Duo Zhang, Leilei Wang, Lu Tang, Yeting Zhang, Huaiyong Zhang, Lin Zou
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引用次数: 0

摘要

衰老与有害的骨质流失有关,往往导致脆性骨折,这可能是由氧化应激引起的。在本研究中,对比幼年、成年和老年C57BL/6J小鼠的差异结果发现,老年小鼠的骨小梁体积明显低于幼年小鼠,骨骼特征与氧化状态显著相关。为了消除衰老的不良影响,将二甲基亚砜的稳定代谢物甲基磺酰甲烷(MSM)添加到73周龄小鼠的饮用水中(400 mg/kg/天),持续8周。MSM的补充改善了胫骨的最大负荷、骨微结构和骨细胞特异性基因的mRNA水平。此外,MSM降低了血清I型胶原c端末端肽(骨吸收的标志)的水平,下调了破骨细胞增殖和活性相关基因的mRNA水平。MSM还降低了血清和骨髓中促炎细胞因子的水平。重要的是,msm处理小鼠表现出增强的抗氧化状态,其特征是血浆、红细胞和骨髓中谷胱甘肽过氧化物酶(GPx)活性和谷胱甘肽浓度增加。这些改善与核因子E2相关因子2 (Nrf2)途径的激活及其下游抗氧化基因表达有关,包括超氧化物歧化酶和GPx。这些发现表明,与年龄相关的骨质流失与氧化应激密切相关,MSM补充剂通过减轻氧化应激介导的骨吸收有效地逆转骨质流失。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

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Protective Effect of Methyl Sulfonyl Methane on the Progression of Age-Induced Bone Loss by Regulating Oxidative Stress-Mediated Bone Resorption.

Aging is associated with detrimental bone loss, often leading to fragility fractures, which may be driven by oxidative stress. In this study, the outcomes of comparing the differences among young, adult and aged C57BL/6J mice found that the trabecular bone volume was significantly lower in the aged mice compared to young mice, and the bone characteristics were significantly correlated with the oxidative status. To counteract the adverse effects of aging, methyl sulfonyl methane (MSM), a stable metabolite of dimethyl sulfoxide, was used to supplement the drinking water (400 mg/kg/day) of the aged mice (73 weeks old) for 8 weeks. The MSM supplementation improved the maximum load, bone microarchitecture, and mRNA levels of osteocyte-specific genes in the tibia. Furthermore, MSM reduced the serum level of the C-terminal telopeptide of type I collagen, a marker of bone resorption, and downregulated the mRNA levels of genes related to osteoclast proliferation and activity. MSM also decreased the levels of pro-inflammatory cytokines in both the serum and bone marrow. Importantly, the MSM-treated mice exhibited an enhanced antioxidant status, characterized by increased glutathione peroxidase (GPx) activity and glutathione concentration in plasma, erythrocytes and bone marrow. These improvements were linked to the activation of the nuclear factor E2 related factor 2 (Nrf2) pathway and its downstream antioxidant gene expression, including that of superoxide dismutase and GPx. These findings suggested that age-related bone loss is closely tied to oxidative stress, and MSM supplementation effectively reverses bone loss by mitigating oxidative stress-mediated bone resorption.

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来源期刊
Antioxidants
Antioxidants Biochemistry, Genetics and Molecular Biology-Physiology
CiteScore
10.60
自引率
11.40%
发文量
2123
审稿时长
16.3 days
期刊介绍: Antioxidants (ISSN 2076-3921), provides an advanced forum for studies related to the science and technology of antioxidants. It publishes research papers, reviews and communications. Our aim is to encourage scientists to publish their experimental and theoretical results in as much detail as possible. There is no restriction on the length of the papers. The full experimental details must be provided so that the results can be reproduced. Electronic files and software regarding the full details of the calculation or experimental procedure, if unable to be published in a normal way, can be deposited as supplementary electronic material.
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