Cystic fibrosis and metabolic alkalosis in children--revisited.

the alkalosis, to a renal K loss via the collecting duct which is partly the reason for the observed hypokalemia. This regulative and physiological hyperaldosteronism, sometimes falsely named ‘pseudo-Bartter’ in this context, in fact supports the generation of a metabolic alkalosis. Therefore, metabolic alkalosis seen in cystic fibrosis patients should not be named ‘pseudo-Bartter’. The term ‘pseudo-Bartter’ should exclusively be used in case of a primary renal involvement mimicking the urinary losses of electrolytes located in the loop of Henle as seen in Bartter’s syndrome. Such a ‘pseudo-Bartter’ would be evident for Dear Sir,