分裂复合体增强子中的删除映射

IF 4.6 Q2 MATERIALS SCIENCE, BIOMATERIALS ACS Applied Bio Materials Pub Date : 2015-01-14 DOI:10.1111/hrd2.00065
Elisa Wurmbach, Anette Preiss
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引用次数: 5

摘要

分裂复合体的增强子[E(spl)-C]由12个不同类别的基因组成。7个基因编码具有碱性螺旋-环-螺旋-橙色(bHLH-O)结构域的蛋白,其功能是转录抑制因子和Notch信号通路的效应器。它们被命名为E(spl)m8-、m7-、m5-、m3-、mβ-、mγ-和mδ-HLH。4个基因E(sp1)m6-、m4-、m2-和mα-BFM相互交织,编码BFM家族的Notch抑制蛋白。该复合体由一个功能不相关的基因分裂,该基因编码kazal型蛋白酶抑制剂(kazal -m1)。一个家族中的所有成员,bHLH-O或BFM,在结构和功能上都非常相似。为了产生特异性突变体,我们分别动员了位于E(spl)m7-HLH和E(spl)m - γ- hlh旁边的p元素构建体。由此产生的缺失被分子和细胞学定位。两个小的缺失只影响E(spl)m7-HLH和E(spl)mδ。有缺陷的果蝇可存活,无明显表型。较大的缺失,也由x射线诱变产生,揭示了大部分E(spl)-C。分析了纯合子缺陷胚胎的表型,以表征各自Notch信号活性的丧失。
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Deletion mapping in the Enhancer of split complex

The Enhancer of split complex [E(spl)-C] comprises twelve genes of different classes. Seven genes encode proteins of with a basic-helix-loop-helix-orange (bHLH-O) domain that function as transcriptional repressors and serve as effectors of the Notch signalling pathway. They have been named E(spl)m8-, m7-, m5-, m3-, mβ-, mγ- and mδ-HLH. Four genes, E(spl)m6-, m4-, m2- and mα-BFM are intermingled and encode Notch repressor proteins of the Bearded-family (BFM). The complex is split by a single gene of unrelated function, encoding a Kazal-type protease inhibitor (Kaz-m1). All members within a family, bHLH-O or BFM, are very similar in structure and in function. In an attempt to generate specific mutants, we have mobilised P-element constructs residing next to E(spl)m7-HLH and E(spl)mγ-HLH, respectively. The resulting deletions were mapped molecularly and by cytology. Two small deletions affected only E(spl)m7-HLH and E(spl)mδ. The deficient flies were viable without apparent phenotype. Larger deletions, generated also by X-ray mutagenesis, uncover most of the E(spl)-C. The phenotypes of homozygous deficient embryos were analysed to characterize the respective loss of Notch signalling activity.

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来源期刊
ACS Applied Bio Materials
ACS Applied Bio Materials Chemistry-Chemistry (all)
CiteScore
9.40
自引率
2.10%
发文量
464
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