干扰脂蛋白成熟使耐甲氧西林金黄色葡萄球菌对人ia组分泌磷脂酶A2和达托霉素增敏。

IF 4.7 3区 医学 Q2 IMMUNOLOGY Journal of Innate Immunity Pub Date : 2023-01-01 Epub Date: 2022-12-06 DOI:10.1159/000527549
Marieke M Kuijk, Yongzheng Wu, Vincent P van Hensbergen, Gizem Shanlitourk, Christine Payré, Gérard Lambeau, Sandra Man-Bovenkerk, Jennifer Herrmann, Rolf Müller, Jos A G van Strijp, Yvonne Pannekoek, Lhousseine Touqui, Nina M van Sorge
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引用次数: 0

摘要

耐甲氧西林金黄色葡萄球菌(MRSA)已被世界卫生组织列为高度优先的病原体,强调了对治疗感染的新疗法的高需求。人iia组分泌的磷脂酶A2 (hGIIA)是对革兰氏阳性细菌(包括金黄色葡萄球菌)最有效的杀菌蛋白之一。为了确定MRSA对hGIIA的抗性机制,我们使用亚致死浓度的重组hGIIA筛选了内布拉斯加转座子突变文库。我们在体外实验和hGIIA转基因小鼠感染模型中发现并证实了编码脂蛋白信号肽酶lspA作为hGIIA新抗性基因的作用。lspA突变体的易感性增加与细胞膜上hGIIA活性增强有关。此外,lspA缺失增加了对达托霉素的易感性,达托霉素是治疗MRSA感染的最后一种抗生素。MRSA野生型可以通过暴露于lspa特异性抑制剂globomycin和myxovirescin A1使hGIIA和达托霉素致敏。对bbb26000金黄色葡萄球菌基因组的分析表明,LspA具有高度的序列保守性,表明LspA抑制具有普遍的应用价值。LspA在hGIIA耐药中的作用并不局限于MRSA,因为在LspA缺失或LspA抑制后,变形链球菌和粪肠球菌也对hGIIA更敏感。总的来说,我们的数据表明,对LspA的药理学干扰可能会解除包括MRSA在内的革兰氏阳性病原体的武装,从而增强先天宿主防御分子和临床应用抗生素的清除能力。
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Interference with Lipoprotein Maturation Sensitizes Methicillin-Resistant Staphylococcus aureus to Human Group IIA-Secreted Phospholipase A2 and Daptomycin.

Methicillin-resistant Staphylococcus aureus (MRSA) has been classified as a high priority pathogen by the World Health Organization underlining the high demand for new therapeutics to treat infections. Human group IIA-secreted phospholipase A2 (hGIIA) is among the most potent bactericidal proteins against Gram-positive bacteria, including S. aureus. To determine hGIIA-resistance mechanisms of MRSA, we screened the Nebraska Transposon Mutant Library using a sublethal concentration of recombinant hGIIA. We identified and confirmed the role of lspA, encoding the lipoprotein signal peptidase LspA, as a new hGIIA resistance gene in both in vitro assays and an infection model in hGIIA-transgenic mice. Increased susceptibility of the lspA mutant was associated with enhanced activity of hGIIA on the cell membrane. Moreover, lspA deletion increased susceptibility to daptomycin, a last-resort antibiotic to treat MRSA infections. MRSA wild type could be sensitized to hGIIA and daptomycin killing through exposure to LspA-specific inhibitors globomycin and myxovirescin A1. Analysis of >26,000 S. aureus genomes showed that LspA is highly sequence-conserved, suggesting universal application of LspA inhibition. The role of LspA in hGIIA resistance was not restricted to MRSA since Streptococcus mutans and Enterococcus faecalis were also more hGIIA-susceptible after lspA deletion or LspA inhibition, respectively. Overall, our data suggest that pharmacological interference with LspA may disarm Gram-positive pathogens, including MRSA, to enhance clearance by innate host defense molecules and clinically applied antibiotics.

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来源期刊
Journal of Innate Immunity
Journal of Innate Immunity 医学-免疫学
CiteScore
10.50
自引率
1.90%
发文量
35
审稿时长
7.5 months
期刊介绍: The ''Journal of Innate Immunity'' is a bimonthly journal covering all aspects within the area of innate immunity, including evolution of the immune system, molecular biology of cells involved in innate immunity, pattern recognition and signals of ‘danger’, microbial corruption, host response and inflammation, mucosal immunity, complement and coagulation, sepsis and septic shock, molecular genomics, and development of immunotherapies. The journal publishes original research articles, short communications, reviews, commentaries and letters to the editors. In addition to regular papers, some issues feature a special section with a thematic focus.
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